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List of works by Martin J Allday

An ATM/Chk2-mediated DNA damage-responsive signaling pathway suppresses Epstein-Barr virus transformation of primary human B cells

scientific article

BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV

scientific article

Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression

scientific article published on 29 November 2016

Correction: Induction of p16INK4a Is the Major Barrier to Proliferation when Epstein-Barr Virus (EBV) Transforms Primary B Cells into Lymphoblastoid Cell Lines.

scientific article published on 5 March 2013

Deregulation of the cell cycle by the Epstein-Barr virus

scientific article

EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors

scientific article published on 12 March 2012

EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells

scientific article published on 11 January 2016

EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency

scientific article

EBV finds a polycomb-mediated, epigenetic solution to the problem of oncogenic stress responses triggered by infection

scientific article published on 24 October 2013

EBV persistence without its EBNA3A and 3C oncogenes in vivo

scientific article published on 30 April 2018

Epigenetic regulation of the latency-associated region of Marek's disease virus in tumor-derived T-cell lines and primary lymphoma.

scientific article

Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP.

scientific article

Epigenetic reprogramming of host genes in viral and microbial pathogenesis

scientific article published on 18 August 2010

Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.

scientific article published on 24 January 2018

Epstein-Barr Virus Nuclear Antigen 3C Inhibits Expression of and the Locus via Interaction with the Histone Lysine Demethylase KDM2B

scientific article published on 12 October 2018

Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.

scientific article published on 8 July 2015

Epstein-Barr virus EBNA3 proteins bind to the C8/alpha7 subunit of the 20S proteasome and are degraded by 20S proteasomes in vitro, but are very stable in latently infected B cells

scientific article

Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection

scientific article

Epstein-Barr virus nuclear antigen (EBNA) 3A induces the expression of and interacts with a subset of chaperones and co-chaperones.

scientific article

Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells

scientific article published on 23 May 2016

Epstein-Barr virus selectively deregulates DNA damage responses in normal B cells but has no detectable effect on regulation of the tumor suppressor p53.

scientific article published on 20 September 2006

Epstein-barr virus latency in B cells leads to epigenetic repression and CpG methylation of the tumour suppressor gene Bim

scientific article published on 26 June 2009

Epstein-barr virus-induced resistance to drugs that activate the mitotic spindle assembly checkpoint in Burkitt's lymphoma cells

scientific article published on 11 October 2006

Epstein–Barr virus can inhibit genotoxin-induced G1 arrest downstream of p53 by preventing the inactivation of CDK2 ⬇️

scientific article published on October 16, 2003

Extensive co-operation between the Epstein-Barr virus EBNA3 proteins in the manipulation of host gene expression and epigenetic chromatin modification

scientific article

Genome diversity of Epstein-Barr virus from multiple tumor types and normal infection

scientific article published on 18 March 2015

Homodimerization of the Meq viral oncoprotein is necessary for induction of T-cell lymphoma by Marek's disease virus

scientific article published on 19 August 2009

How does Epstein-Barr virus (EBV) complement the activation of Myc in the pathogenesis of Burkitt's lymphoma?

scientific article published on 25 July 2009

Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines

scientific article

Interaction of MEQ protein and C-terminal-binding protein is critical for induction of lymphomas by Marek's disease virus

scholarly article

Latent Epstein-Barr virus can inhibit apoptosis in B cells by blocking the induction of NOXA expression

scientific article

MDM2 promotes proteasome-dependent ubiquitin-independent degradation of retinoblastoma protein.

scientific article published on December 2005

Mutagenesis of the herpesvirus saimiri terminal repeat region reveals important elements for virus production

scientific article published on 11 April 2007

Persistent KSHV Infection Increases EBV-Associated Tumor Formation In Vivo via Enhanced EBV Lytic Gene Expression.

scientific article published on July 2017

Proliferation and differentiation in isogenic populations of peripheral B cells activated by Epstein-Barr virus or T cell-derived mitogens

scientific article published in April 2004

Requirement for PRC1 subunit BMI1 in host gene activation by Epstein-Barr virus protein EBNA3C

article

The EBNA3 Family: Two Oncoproteins and a Tumour Suppressor that Are Central to the Biology of EBV in B Cells

scientific article published on January 2015

The curious case of the tumour virus: 50 years of Burkitt's lymphoma

scientific article published on December 2008

The transcriptional co-repressor C-terminal binding protein (CtBP) associates with centrosomes during mitosis

scientific article published in March 2006

Transcriptional silencing of Polo-like kinase 2 (SNK/PLK2) is a frequent event in B-cell malignancies.

scientific article published on 13 September 2005

Two nonconsensus sites in the Epstein-Barr virus oncoprotein EBNA3A cooperate to bind the co-repressor carboxyl-terminal-binding protein (CtBP)

scientific journal article

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