List of works - Wei-Chieh Chiang

ATF6 is essential for human cone photoreceptor development

scientific article published in 2021

Ablation of Chop Transiently Enhances Photoreceptor Survival but Does Not Prevent Retinal Degeneration in Transgenic Mice Expressing Human P23H Rhodopsin

scientific article published in January 2016

Achromatopsia mutations target sequential steps of ATF6 activation.

scientific article published on 27 December 2016

Autosomal recessive cone-rod dystrophy can be caused by mutations in the ATF6 gene.

scientific article published on 16 August 2017

ER stress and unfolded protein response in ocular health and disease

scientific article published on 20 June 2018

Endoplasmic Reticulum-Associated Degradation (ERAD) of Misfolded Glycoproteins and Mutant P23H Rhodopsin in Photoreceptor Cells ⬇️

scientific article published on January 1, 2012

Endoplasmic reticulum stress in human photoreceptor diseases.

scientific article published on 23 April 2016

Enzymatic and transcriptional regulation of human ecto-ATPase/E-NTPDase 2 ⬇️

scientific article published on October 15, 2003

IRE1 directs proteasomal and lysosomal degradation of misfolded rhodopsin ⬇️

scientific article published on January 4, 2012

In Vivo Visualization of Endoplasmic Reticulum Stress in the Retina Using the ERAI Reporter Mouse

scientific article

Induction of endoplasmic reticulum stress genes, BiP and chop, in genetic and environmental models of retinal degeneration

scientific article

Inhibition of human NTPDase 2 by modification of an intramembrane cysteine by p-chloromercuriphenylsulfonate and oxidative cross-linking of the transmembrane domains.

scientific article

Multiple Mechanisms of Unfolded Protein Response-Induced Cell Death

scientific article published on 05 May 2015

Mutations in the unfolded protein response regulator ATF6 cause the cone dysfunction disorder achromatopsia

scientific journal article

Robust Endoplasmic Reticulum-Associated Degradation of Rhodopsin Precedes Retinal Degeneration.

scientific article published on 2 October 2014

Selective activation of ATF6 and PERK endoplasmic reticulum stress signaling pathways prevent mutant rhodopsin accumulation

scientific article

The unfolded protein response regulator ATF6 promotes mesodermal differentiation.

scientific article published on 13 February 2018

List of works by Wei-Chieh Chiang

ATF6 is essential for human cone photoreceptor development

Ablation of Chop Transiently Enhances Photoreceptor Survival but Does Not Prevent Retinal Degeneration in Transgenic Mice Expressing Human P23H Rhodopsin

Achromatopsia mutations target sequential steps of ATF6 activation.

Autosomal recessive cone-rod dystrophy can be caused by mutations in the ATF6 gene.

ER stress and unfolded protein response in ocular health and disease

Endoplasmic Reticulum-Associated Degradation (ERAD) of Misfolded Glycoproteins and Mutant P23H Rhodopsin in Photoreceptor Cells ⬇️

Endoplasmic reticulum stress in human photoreceptor diseases.

Enzymatic and transcriptional regulation of human ecto-ATPase/E-NTPDase 2 ⬇️

IRE1 directs proteasomal and lysosomal degradation of misfolded rhodopsin ⬇️

In Vivo Visualization of Endoplasmic Reticulum Stress in the Retina Using the ERAI Reporter Mouse

Induction of endoplasmic reticulum stress genes, BiP and chop, in genetic and environmental models of retinal degeneration

Inhibition of human NTPDase 2 by modification of an intramembrane cysteine by p-chloromercuriphenylsulfonate and oxidative cross-linking of the transmembrane domains.

Multiple Mechanisms of Unfolded Protein Response-Induced Cell Death

Mutations in the unfolded protein response regulator ATF6 cause the cone dysfunction disorder achromatopsia

Robust Endoplasmic Reticulum-Associated Degradation of Rhodopsin Precedes Retinal Degeneration.

Selective activation of ATF6 and PERK endoplasmic reticulum stress signaling pathways prevent mutant rhodopsin accumulation

The unfolded protein response regulator ATF6 promotes mesodermal differentiation.