List of works - Anaïs Briot

A ligand-independent VEGFR2 signaling pathway limits angiogenic responses in diabetes

scientific article

Blockade of specific NOTCH ligands: a new promising approach in cancer therapy

scientific article

Effects of the amino acid derivatives, β-hydroxy-β-methylbutyrate, taurine, and N-methyltyramine, on triacylglycerol breakdown in fat cells

scientific article published on 27 March 2019

Elastase 2 is expressed in human and mouse epidermis and impairs skin barrier function in Netherton syndrome through filaggrin and lipid misprocessing

scientific article

Endothelial NOTCH1 is suppressed by circulating lipids and antagonizes inflammation during atherosclerosis

scientific article

Infliximab Infusions for Netherton Syndrome: Sustained Clinical Improvement Correlates with a Reduction of Thymic Stromal Lymphopoietin Levels in the Skin

scientific article published on 09 June 2011

Lobular architecture of human adipose tissue defines the niche and fate of progenitor cells.

scientific article

NOTCH1 is a mechanosensor in adult arteries

scientific article

Notch, lipids, and endothelial cells.

scientific article published on 22 July 2016

Par2 inactivation inhibits early production of TSLP, but not cutaneous inflammation, in Netherton syndrome adult mouse model.

scientific article published on 12 August 2010

Pterostilbene Inhibits Lipogenic Activity similar to Resveratrol or Caffeine but Differently Modulates Lipolysis in Adipocytes

scientific article published on 19 June 2017

Repression of Sox9 by Jag1 is continuously required to suppress the default chondrogenic fate of vascular smooth muscle cells

scientific article

Senescence Alters PPARG (Peroxisome Proliferator-Activated Receptor Gamma)-Dependent Fatty Acid Handling in Human Adipose Tissue Microvascular Endothelial Cells and Favors Inflammation.

scientific article published on 15 March 2018

Sox17 is indispensable for acquisition and maintenance of arterial identity

scientific article

Testosterone levels influence mouse fetal Leydig cell progenitors through notch signaling.

scientific article

Transgenic kallikrein 5 mice reproduce major cutaneous and systemic hallmarks of Netherton syndrome.

scientific article published on 17 February 2014

List of works by Anaïs Briot

A ligand-independent VEGFR2 signaling pathway limits angiogenic responses in diabetes

Blockade of specific NOTCH ligands: a new promising approach in cancer therapy

Effects of the amino acid derivatives, β-hydroxy-β-methylbutyrate, taurine, and N-methyltyramine, on triacylglycerol breakdown in fat cells

Elastase 2 is expressed in human and mouse epidermis and impairs skin barrier function in Netherton syndrome through filaggrin and lipid misprocessing

Endothelial NOTCH1 is suppressed by circulating lipids and antagonizes inflammation during atherosclerosis

Infliximab Infusions for Netherton Syndrome: Sustained Clinical Improvement Correlates with a Reduction of Thymic Stromal Lymphopoietin Levels in the Skin

Lobular architecture of human adipose tissue defines the niche and fate of progenitor cells.

NOTCH1 is a mechanosensor in adult arteries

Notch, lipids, and endothelial cells.

Par2 inactivation inhibits early production of TSLP, but not cutaneous inflammation, in Netherton syndrome adult mouse model.

Pterostilbene Inhibits Lipogenic Activity similar to Resveratrol or Caffeine but Differently Modulates Lipolysis in Adipocytes

Repression of Sox9 by Jag1 is continuously required to suppress the default chondrogenic fate of vascular smooth muscle cells

Senescence Alters PPARG (Peroxisome Proliferator-Activated Receptor Gamma)-Dependent Fatty Acid Handling in Human Adipose Tissue Microvascular Endothelial Cells and Favors Inflammation.

Sox17 is indispensable for acquisition and maintenance of arterial identity

Testosterone levels influence mouse fetal Leydig cell progenitors through notch signaling.

Transgenic kallikrein 5 mice reproduce major cutaneous and systemic hallmarks of Netherton syndrome.