List of works - Amy S Guala

Absence of c-Jun NH2-terminal kinase 1 protects against house dust mite-induced pulmonary remodeling but not airway hyperresponsiveness and inflammation

scientific article

Cigarette smoke targets glutaredoxin 1, increasing s-glutathionylation and epithelial cell death.

scientific article published on 31 March 2011

Distinct functions of airway epithelial nuclear factor-kappaB activity regulate nitrogen dioxide-induced acute lung injury

scientific article published on 9 November 2009

Genetic ablation of glutaredoxin-1 causes enhanced resolution of airways hyperresponsiveness and mucus metaplasia in mice with allergic airways disease ⬇️

scientific article published on June 29, 2012

Glutaredoxin-1 attenuates S-glutathionylation of the death receptor fas and decreases resolution of Pseudomonas aeruginosa pneumonia.

scientific article published on February 2014

In situ analysis of protein S-glutathionylation in lung tissue using glutaredoxin-1-catalyzed cysteine derivatization.

scientific article published on July 2009

In situ detection of S-glutathionylated proteins following glutaredoxin-1 catalyzed cysteine derivatization.

scientific article published on 20 February 2006

Induction of a mesenchymal expression program in lung epithelial cells by wingless protein (Wnt)/β-catenin requires the presence of c-Jun N-terminal kinase-1 (JNK1).

scientific article

Influenza Induces Endoplasmic Reticulum Stress, Caspase-12–Dependent Apoptosis, and c-Jun N-Terminal Kinase–Mediated Transforming Growth Factor–β Release in Lung Epithelial Cells ⬇️

scientific article published on July 28, 2011

Nitric oxide represses inhibitory kappaB kinase through S-nitrosylation

scholarly article

Nuclear factor-kappaB activation in airway epithelium induces inflammation and hyperresponsiveness

scientific article published on 8 February 2008

Reactive nitrogen species-induced cell death requires Fas-dependent activation of c-Jun N-terminal kinase

scientific article published on August 2004

Redox-based regulation of apoptosis: S-glutathionylation as a regulatory mechanism to control cell death

scientific article

Regulation of apoptosis through cysteine oxidation: implications for fibrotic lung disease.

scientific article published on August 2010

Strain-dependent activation of NF-kappaB in the airway epithelium and its role in allergic airway inflammation

scientific article

The glutaredoxin/S-glutathionylation axis regulates interleukin-17A-induced proinflammatory responses in lung epithelial cells in association with S-glutathionylation of nuclear factor κB family proteins

scientific article

c-Jun N-terminal kinase 1 promotes transforming growth factor-β1-induced epithelial-to-mesenchymal transition via control of linker phosphorylation and transcriptional activity of Smad3.

scientific article published on 25 June 2010

List of works by Amy S Guala

Absence of c-Jun NH2-terminal kinase 1 protects against house dust mite-induced pulmonary remodeling but not airway hyperresponsiveness and inflammation

Cigarette smoke targets glutaredoxin 1, increasing s-glutathionylation and epithelial cell death.

Distinct functions of airway epithelial nuclear factor-kappaB activity regulate nitrogen dioxide-induced acute lung injury

Genetic ablation of glutaredoxin-1 causes enhanced resolution of airways hyperresponsiveness and mucus metaplasia in mice with allergic airways disease ⬇️

Glutaredoxin-1 attenuates S-glutathionylation of the death receptor fas and decreases resolution of Pseudomonas aeruginosa pneumonia.

In situ analysis of protein S-glutathionylation in lung tissue using glutaredoxin-1-catalyzed cysteine derivatization.

In situ detection of S-glutathionylated proteins following glutaredoxin-1 catalyzed cysteine derivatization.

Induction of a mesenchymal expression program in lung epithelial cells by wingless protein (Wnt)/β-catenin requires the presence of c-Jun N-terminal kinase-1 (JNK1).

Influenza Induces Endoplasmic Reticulum Stress, Caspase-12–Dependent Apoptosis, and c-Jun N-Terminal Kinase–Mediated Transforming Growth Factor–β Release in Lung Epithelial Cells ⬇️

Nitric oxide represses inhibitory kappaB kinase through S-nitrosylation

Nuclear factor-kappaB activation in airway epithelium induces inflammation and hyperresponsiveness

Reactive nitrogen species-induced cell death requires Fas-dependent activation of c-Jun N-terminal kinase

Redox-based regulation of apoptosis: S-glutathionylation as a regulatory mechanism to control cell death

Regulation of apoptosis through cysteine oxidation: implications for fibrotic lung disease.

Strain-dependent activation of NF-kappaB in the airway epithelium and its role in allergic airway inflammation

The glutaredoxin/S-glutathionylation axis regulates interleukin-17A-induced proinflammatory responses in lung epithelial cells in association with S-glutathionylation of nuclear factor κB family proteins

c-Jun N-terminal kinase 1 promotes transforming growth factor-β1-induced epithelial-to-mesenchymal transition via control of linker phosphorylation and transcriptional activity of Smad3.