List of works - Andrea N Ladd

Alternative splicing of the neurofibromatosis type 1 pre-mRNA is regulated by the muscleblind-like proteins and the CUG-BP and ELAV-like factors

scientific article published on 10 December 2012

Andrea Ladd: Getting to the heart of alternative splicing control. Interview by Ruth Williams.

scientific article published on December 2008

CELF6, a member of the CELF family of RNA-binding proteins, regulates muscle-specific splicing enhancer-dependent alternative splicing

scientific article

CUG-BP, Elav-like family (CELF)-mediated alternative splicing regulation in the brain during health and disease.

scientific article published on 14 December 2012

Cardiac tissue-specific repression of CELF activity disrupts alternative splicing and causes cardiomyopathy.

scientific article

Cloning and embryonic expression patterns of the chicken CELF family.

scientific article published in August 2007

Differential susceptibility of muscles to myotonia and force impairment in a mouse model of myotonic dystrophy.

scientific article published on 14 March 2011

Expression of a dominant negative CELF protein in vivo leads to altered muscle organization, fiber size, and subtype.

scientific article

Finding signals that regulate alternative splicing in the post-genomic era

scientific article

Gene expression analyses implicate an alternative splicing program in regulating contractile gene expression and serum response factor activity in mice

scientific article

Known turnover and translation regulatory RNA-binding proteins interact with the 3' UTR of SECIS-binding protein 2.

scientific article published on 05 January 2009

Loss of muscleblind-like 1 promotes invasive mesenchyme formation in endocardial cushions by stimulating autocrine TGFβ3.

scientific article

Muscleblind-like 1 is a negative regulator of TGF-beta-dependent epithelial-mesenchymal transition of atrioventricular canal endocardial cells.

scientific article

Repression of nuclear CELF activity can rescue CELF-regulated alternative splicing defects in skeletal muscle models of myotonic dystrophy

scientific article

The importance of CELF control: molecular and biological roles of the CUG‐BP, Elav‐like family of RNA‐binding proteins ⬇️

scientific article published on August 17, 2011

The neurofibromatosis type I pre-mRNA is a novel target of CELF protein-mediated splicing regulation

scientific article

List of works by Andrea N Ladd

Alternative splicing of the neurofibromatosis type 1 pre-mRNA is regulated by the muscleblind-like proteins and the CUG-BP and ELAV-like factors

Andrea Ladd: Getting to the heart of alternative splicing control. Interview by Ruth Williams.

CELF6, a member of the CELF family of RNA-binding proteins, regulates muscle-specific splicing enhancer-dependent alternative splicing

CUG-BP, Elav-like family (CELF)-mediated alternative splicing regulation in the brain during health and disease.

Cardiac tissue-specific repression of CELF activity disrupts alternative splicing and causes cardiomyopathy.

Cloning and embryonic expression patterns of the chicken CELF family.

Differential susceptibility of muscles to myotonia and force impairment in a mouse model of myotonic dystrophy.

Expression of a dominant negative CELF protein in vivo leads to altered muscle organization, fiber size, and subtype.

Finding signals that regulate alternative splicing in the post-genomic era

Gene expression analyses implicate an alternative splicing program in regulating contractile gene expression and serum response factor activity in mice

Known turnover and translation regulatory RNA-binding proteins interact with the 3' UTR of SECIS-binding protein 2.

Loss of muscleblind-like 1 promotes invasive mesenchyme formation in endocardial cushions by stimulating autocrine TGFβ3.

Muscleblind-like 1 is a negative regulator of TGF-beta-dependent epithelial-mesenchymal transition of atrioventricular canal endocardial cells.

Repression of nuclear CELF activity can rescue CELF-regulated alternative splicing defects in skeletal muscle models of myotonic dystrophy

The importance of CELF control: molecular and biological roles of the CUG‐BP, Elav‐like family of RNA‐binding proteins ⬇️

The neurofibromatosis type I pre-mRNA is a novel target of CELF protein-mediated splicing regulation