List of works - Luís Maia

A case of haemophagocytic syndrome presenting with oculogyric crises

scientific article published on 01 April 2010

Adapter dimer contamination in sRNA‐sequencing datasets predicts sequencing failure and batch effects and hampers extracellular vesicle‐sRNA analysis

scientific article published in 2023

CNS involvement in V30M transthyretin amyloidosis: clinical, neuropathological and biochemical findings

scientific article published on 4 August 2014

Clinical phenotypes of Cerebral Amyloid Angiopathy.

scientific article published on 6 March 2007

Commentary on 'Subcortical hematoma caused by cerebral amyloid angiopathy: does the first evidence of hemorrhage occur in the subarachnoid space?' (Neuropathology 2003; 23, 254-261)

scientific article published on 01 December 2004

Efficiency of silencing RNA for removal of transthyretin V30M in a TTR leptomeningeal animal model

scientific article published on 24 November 2016

Emerging CNS involvement in FAP-TTR long survival patients.

scientific article published on 2 November 2015

Evidence for a specific intracellular localization of an antisense oligonucleotide in k562 cells.

scientific article published on September 2005

Lobar brain hemorrhages and white matter changes: Clinical, radiological and laboratorial profiles.

scientific article

Motor, cognitive and mobility deficits in 1000 geriatric patients: protocol of a quantitative observational study before and after routine clinical geriatric treatment - the ComOn-study

scientific article published on 06 February 2020

Oral anticoagulation in patients with "hemorrhagic-prone" vasculopathies: Overestimated risk?

scientific article published on 19 April 2019

Physiologically regulated transgenic ABCA1 does not reduce amyloid burden or amyloid-beta peptide levels in vivo.

scientific article

Plasma neurofilament light chain: an early biomarker for hereditary ATTR amyloid polyneuropathy

scientific article published on 06 January 2020

Recurrent cerebrospinal fluid escape in an HIV-1-infected patient receiving antiretroviral therapy

scientific article published in April 2016

Superficial siderosis: a potential diagnostic marker of cerebral amyloid angiopathy in Alzheimer disease.

scientific article

The absence of ABCA1 decreases soluble ApoE levels but does not diminish amyloid deposition in two murine models of Alzheimer disease.

scientific article

List of works by Luís Maia

A case of haemophagocytic syndrome presenting with oculogyric crises

Adapter dimer contamination in sRNA‐sequencing datasets predicts sequencing failure and batch effects and hampers extracellular vesicle‐sRNA analysis

CNS involvement in V30M transthyretin amyloidosis: clinical, neuropathological and biochemical findings

Clinical phenotypes of Cerebral Amyloid Angiopathy.

Commentary on 'Subcortical hematoma caused by cerebral amyloid angiopathy: does the first evidence of hemorrhage occur in the subarachnoid space?' (Neuropathology 2003; 23, 254-261)

Efficiency of silencing RNA for removal of transthyretin V30M in a TTR leptomeningeal animal model

Emerging CNS involvement in FAP-TTR long survival patients.

Evidence for a specific intracellular localization of an antisense oligonucleotide in k562 cells.

Lobar brain hemorrhages and white matter changes: Clinical, radiological and laboratorial profiles.

Motor, cognitive and mobility deficits in 1000 geriatric patients: protocol of a quantitative observational study before and after routine clinical geriatric treatment - the ComOn-study

Oral anticoagulation in patients with "hemorrhagic-prone" vasculopathies: Overestimated risk?

Physiologically regulated transgenic ABCA1 does not reduce amyloid burden or amyloid-beta peptide levels in vivo.

Plasma neurofilament light chain: an early biomarker for hereditary ATTR amyloid polyneuropathy

Recurrent cerebrospinal fluid escape in an HIV-1-infected patient receiving antiretroviral therapy

Superficial siderosis: a potential diagnostic marker of cerebral amyloid angiopathy in Alzheimer disease.

The absence of ABCA1 decreases soluble ApoE levels but does not diminish amyloid deposition in two murine models of Alzheimer disease.