List of works - Nicola J Lee

Amygdala NPY Circuits Promote the Development of Accelerated Obesity under Chronic Stress Conditions

scientific article published on 25 April 2019

Central RANK signalling in NPY neurons alters bone mass in male mice

scientific article published on 15 February 2018

Compensatory changes in [125I]-PYY binding in Y receptor knockout mice suggest the potential existence of further Y receptor(s)

scientific article published in February 2005

Double deletion of orexigenic neuropeptide Y and dynorphin results in paradoxical obesity in mice

scientific article

Energy partitioning between fat and bone mass is controlled via a hypothalamic leptin/NPY relay

scientific article published on 09 March 2020

Lack of NPY in neurotensin neurons leads to a lean phenotype

scientific article published on 12 November 2019

NPY regulation of bone remodelling.

scientific article published on 11 September 2009

NPY signalling in early osteoblasts controls glucose homeostasis.

scientific article

Neuropeptide Y knockout mice reveal a central role of NPY in the coordination of bone mass to body weight

scientific article

Novel role of Y1 receptors in the coordinated regulation of bone and energy homeostasis

scientific article

Osteoblast specific Y1 receptor deletion enhances bone mass

scientific article published in March 2011

Peptide YY ablation in mice leads to the development of hyperinsulinaemia and obesity

scientific article

Peripheral neuropeptide Y Y1 receptors regulate lipid oxidation and fat accretion

scientific article published on 17 November 2009

Prader-Willi Critical Region, a Non-Translated, Imprinted Central Regulator of Bone Mass: Possible Role in Skeletal Abnormalities in Prader-Willi Syndrome.

scientific article published on 29 January 2016

RANK deletion in neuropeptide Y neurones attenuates oestrogen deficiency-related bone loss

scientific article published on 01 February 2019

Snord116 is critical in the regulation of food intake and body weight

scientific article

The y6 receptor suppresses bone resorption and stimulates bone formation in mice via a suprachiasmatic nucleus relay.

scientific article published on 22 December 2015

Tumor-induced anorexia and weight loss are mediated by the TGF-beta superfamily cytokine MIC-1.

scientific article published on 4 November 2007

Uncoupling protein-1 is protective of bone mass under mild cold stress conditions.

scientific article published on 6 June 2015

List of works by Nicola J Lee

Amygdala NPY Circuits Promote the Development of Accelerated Obesity under Chronic Stress Conditions

Central RANK signalling in NPY neurons alters bone mass in male mice

Compensatory changes in [125I]-PYY binding in Y receptor knockout mice suggest the potential existence of further Y receptor(s)

Double deletion of orexigenic neuropeptide Y and dynorphin results in paradoxical obesity in mice

Energy partitioning between fat and bone mass is controlled via a hypothalamic leptin/NPY relay

Lack of NPY in neurotensin neurons leads to a lean phenotype

NPY regulation of bone remodelling.

NPY signalling in early osteoblasts controls glucose homeostasis.

Neuropeptide Y knockout mice reveal a central role of NPY in the coordination of bone mass to body weight

Novel role of Y1 receptors in the coordinated regulation of bone and energy homeostasis

Osteoblast specific Y1 receptor deletion enhances bone mass

Peptide YY ablation in mice leads to the development of hyperinsulinaemia and obesity

Peripheral neuropeptide Y Y1 receptors regulate lipid oxidation and fat accretion

Prader-Willi Critical Region, a Non-Translated, Imprinted Central Regulator of Bone Mass: Possible Role in Skeletal Abnormalities in Prader-Willi Syndrome.

RANK deletion in neuropeptide Y neurones attenuates oestrogen deficiency-related bone loss

Snord116 is critical in the regulation of food intake and body weight

The y6 receptor suppresses bone resorption and stimulates bone formation in mice via a suprachiasmatic nucleus relay.

Tumor-induced anorexia and weight loss are mediated by the TGF-beta superfamily cytokine MIC-1.

Uncoupling protein-1 is protective of bone mass under mild cold stress conditions.