List of works - Alexander M Kleschevnikov

Afferent high strength tetanizations favour potentiation of the NMDA vs. AMPA receptor-mediated component of field EPSP in CA1 hippocampal slices of rats

scientific article published on 01 June 2000

Atropine suppresses associative LTP in the CA1 region of rat hippocampal slices

scientific article published on 01 February 1995

Behavioral parameters of the spatial memory correlate with the potentiation of the population spike, but not with the population excitatory postsynaptic potential, of the CA1 region in rat hippocampal slices.

scientific article

Changes in paired-pulse facilitation correlate with induction of long-term potentiation in area CA1 of rat hippocampal slices.

scientific article published in February 1997

Deficits in cognition and synaptic plasticity in a mouse model of Down syndrome ameliorated by GABAB receptor antagonists.

scientific article

Discoveries in Down syndrome: moving basic science to clinical care.

scientific article published on January 2012

Effects of individual segmental trisomies of human chromosome 21 syntenic regions on hippocampal long-term potentiation and cognitive behaviors in mice.

scientific article

Fimbria-fornix lesions impair spatial performance and induce epileptic-like activity but do not affect long-term potentiation in the CA1 region of rat hippocampal slices.

scientific article published in September 1994

Genetic analysis of Down syndrome facilitated by mouse chromosome engineering.

scientific article published on January 2012

Graphene biointerfaces for optical stimulation of cells.

scientific article published on 18 May 2018

Heterosynaptic short-term depression of population spike amplitude in the pyramidal layer of the CA1 hippocampal region evoked by a theta-like tetanization.

scientific article published in May 1993

Hippocampal long-term potentiation suppressed by increased inhibition in the Ts65Dn mouse, a genetic model of Down syndrome.

scientific article

Human chromosome 21 orthologous region on mouse chromosome 17 is a major determinant of Down syndrome-related developmental cognitive deficits

scientific article published on 16 September 2013

Increased App expression in a mouse model of Down's syndrome disrupts NGF transport and causes cholinergic neuron degeneration.

scientific article

Increased efficiency of the GABAA and GABAB receptor-mediated neurotransmission in the Ts65Dn mouse model of Down syndrome

scientific article published on 17 October 2011

Mouse models for Down syndrome-associated developmental cognitive disabilities

scientific article published on 25 August 2011

Neurophysiological analysis of long-term potentiation in mammalian brain.

scientific article published on January 1995

Restoration of norepinephrine-modulated contextual memory in a mouse model of Down syndrome.

scientific article published in November 2009

The "Down syndrome critical region" is sufficient in the mouse model to confer behavioral, neurophysiological, and synaptic phenotypes characteristic of Down syndrome

scientific article published on May 2009

The M1 antagonist pirenzepine suppresses heterosynaptic STD in the CA1 region

scientific article published on 01 August 1994

Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice

scientific article published on 03 March 2014

List of works by Alexander M Kleschevnikov

Afferent high strength tetanizations favour potentiation of the NMDA vs. AMPA receptor-mediated component of field EPSP in CA1 hippocampal slices of rats

Atropine suppresses associative LTP in the CA1 region of rat hippocampal slices

Behavioral parameters of the spatial memory correlate with the potentiation of the population spike, but not with the population excitatory postsynaptic potential, of the CA1 region in rat hippocampal slices.

Changes in paired-pulse facilitation correlate with induction of long-term potentiation in area CA1 of rat hippocampal slices.

Deficits in cognition and synaptic plasticity in a mouse model of Down syndrome ameliorated by GABAB receptor antagonists.

Discoveries in Down syndrome: moving basic science to clinical care.

Effects of individual segmental trisomies of human chromosome 21 syntenic regions on hippocampal long-term potentiation and cognitive behaviors in mice.

Fimbria-fornix lesions impair spatial performance and induce epileptic-like activity but do not affect long-term potentiation in the CA1 region of rat hippocampal slices.

Genetic analysis of Down syndrome facilitated by mouse chromosome engineering.

Graphene biointerfaces for optical stimulation of cells.

Heterosynaptic short-term depression of population spike amplitude in the pyramidal layer of the CA1 hippocampal region evoked by a theta-like tetanization.

Hippocampal long-term potentiation suppressed by increased inhibition in the Ts65Dn mouse, a genetic model of Down syndrome.

Human chromosome 21 orthologous region on mouse chromosome 17 is a major determinant of Down syndrome-related developmental cognitive deficits

Increased App expression in a mouse model of Down's syndrome disrupts NGF transport and causes cholinergic neuron degeneration.

Increased efficiency of the GABAA and GABAB receptor-mediated neurotransmission in the Ts65Dn mouse model of Down syndrome

Mouse models for Down syndrome-associated developmental cognitive disabilities

Neurophysiological analysis of long-term potentiation in mammalian brain.

Restoration of norepinephrine-modulated contextual memory in a mouse model of Down syndrome.

The "Down syndrome critical region" is sufficient in the mouse model to confer behavioral, neurophysiological, and synaptic phenotypes characteristic of Down syndrome

The M1 antagonist pirenzepine suppresses heterosynaptic STD in the CA1 region

Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice