List of works - Robert Guzy

Comprehensive Care of the Lung Transplant Patient

scientific article published on 8 October 2016

Effect of FGF/FGFR pathway blocking on lung adenocarcinoma and its cancer-associated fibroblasts

scientific article published on 24 June 2019

FGFR2 is Required for AEC2 Homeostasis and Survival Following Bleomycin-Induced Lung Injury

scientific article published on 20 December 2019

Fibroblast Growth Factors Inhibitors in Lung Fibrosis: Friends or Foes?

scientific article published on 05 May 2020

Fibroblast growth factor 2 decreases bleomycin-induced pulmonary fibrosis and inhibits fibroblast collagen production and myofibroblast differentiation

scientific article published on 05 July 2018

Fibroblast growth factor 2 is required for epithelial recovery, but not for pulmonary fibrosis, in response to bleomycin

scientific article

Increases in mitochondrial reactive oxygen species trigger hypoxia-induced calcium responses in pulmonary artery smooth muscle cells

article

Loss of the SdhB, but Not the SdhA, subunit of complex II triggers reactive oxygen species-dependent hypoxia-inducible factor activation and tumorigenesis.

scientific article published on 29 October 2007

Mitochondrial complex III is required for hypoxia-induced ROS production and gene transcription in yeast.

scientific article published in September 2007

Mitochondrial dysfunction resulting from loss of cytochrome c impairs cellular oxygen sensing and hypoxic HIF-alpha activation

scientific article

Mitochondrial oxidant stress triggers cell death in simulated ischemia-reperfusion.

scientific article published on 23 December 2010

Nitric oxide during ischemia attenuates oxidant stress and cell death during ischemia and reperfusion in cardiomyocytes

scientific article published on 18 May 2007

Oxidant stress during simulated ischemia primes cardiomyocytes for cell death during reperfusion.

scientific article published on 7 May 2007

Oxygen sensing by mitochondria at complex III: the paradox of increased reactive oxygen species during hypoxia.

scientific article published on 20 July 2006

Pulmonary fibrosis requires cell-autonomous mesenchymal fibroblast growth factor (FGF) signaling.

scientific article published on 9 May 2017

Superoxide generated at mitochondrial complex III triggers acute responses to hypoxia in the pulmonary circulation.

scientific article published on 17 January 2013

YKL-40 in pediatric asthma, MUC5B promoter polymorphism in idiopathic pulmonary fibrosis, and esmolol in septic shock

scientific article

List of works by Robert Guzy

Comprehensive Care of the Lung Transplant Patient

Effect of FGF/FGFR pathway blocking on lung adenocarcinoma and its cancer-associated fibroblasts

FGFR2 is Required for AEC2 Homeostasis and Survival Following Bleomycin-Induced Lung Injury

Fibroblast Growth Factors Inhibitors in Lung Fibrosis: Friends or Foes?

Fibroblast growth factor 2 decreases bleomycin-induced pulmonary fibrosis and inhibits fibroblast collagen production and myofibroblast differentiation

Fibroblast growth factor 2 is required for epithelial recovery, but not for pulmonary fibrosis, in response to bleomycin

Increases in mitochondrial reactive oxygen species trigger hypoxia-induced calcium responses in pulmonary artery smooth muscle cells

Loss of the SdhB, but Not the SdhA, subunit of complex II triggers reactive oxygen species-dependent hypoxia-inducible factor activation and tumorigenesis.

Mitochondrial complex III is required for hypoxia-induced ROS production and gene transcription in yeast.

Mitochondrial dysfunction resulting from loss of cytochrome c impairs cellular oxygen sensing and hypoxic HIF-alpha activation

Mitochondrial oxidant stress triggers cell death in simulated ischemia-reperfusion.

Nitric oxide during ischemia attenuates oxidant stress and cell death during ischemia and reperfusion in cardiomyocytes

Oxidant stress during simulated ischemia primes cardiomyocytes for cell death during reperfusion.

Oxygen sensing by mitochondria at complex III: the paradox of increased reactive oxygen species during hypoxia.

Pulmonary fibrosis requires cell-autonomous mesenchymal fibroblast growth factor (FGF) signaling.

Superoxide generated at mitochondrial complex III triggers acute responses to hypoxia in the pulmonary circulation.

YKL-40 in pediatric asthma, MUC5B promoter polymorphism in idiopathic pulmonary fibrosis, and esmolol in septic shock