List of works - Alberto Ferri

Acute focal brain damage alters mitochondrial dynamics and autophagy in axotomized neurons.

scientific article

Apaf1 mediates apoptosis and mitochondrial damage induced by mutant human SOD1s typical of familial amyotrophic lateral sclerosis

scientific article

Apoptosome inactivation rescues proneural and neural cells from neurodegeneration.

scientific article published in November 2004

CNF1 Enhances Brain Energy Content and Counteracts Spontaneous Epileptiform Phenomena in Aged DBA/2J Mice

scientific article

CNF1 increases brain energy level, counteracts neuroinflammatory markers and rescues cognitive deficits in a murine model of Alzheimer's disease

scientific article

Calcineurin activity is regulated both by redox compounds and by mutant familial amyotrophic lateral sclerosis-superoxide dismutase

scientific article published on 01 August 2000

Caspase-3 triggers early synaptic dysfunction in a mouse model of Alzheimer's disease

scientific article published on 12 December 2010

Cell death in amyotrophic lateral sclerosis: interplay between neuronal and glial cells.

scientific article published on 18 June 2004

Copper depletion increases the mitochondrial-associated SOD1 in neuronal cells

scientific article published on 8 December 2010

Cysteine 111 affects aggregation and cytotoxicity of mutant Cu,Zn-superoxide dismutase associated with familial amyotrophic lateral sclerosis

scientific article

Different polymeric forms of actin detected by the fluorescent probe terbium ion ⬇️

scientific article published on October 27, 1981

Dynamic NAD(P)H post-synaptic autofluorescence signals for the assessment of mitochondrial function in a neurodegenerative disease: monitoring the primary motor cortex of G93A mice, an amyotrophic lateral sclerosis model.

scientific article published on 10 November 2009

Familial ALS-superoxide dismutases associate with mitochondria and shift their redox potentials.

scholarly article

G-actin modified by plasma membrane interaction polymerizes only in the presence of filamentous myosin

scientific article published on 01 March 1980

Glutaredoxin 1 is a major player in copper metabolism in neuroblastoma cells.

scientific article published on 13 September 2013

Glutaredoxin 2 prevents aggregation of mutant SOD1 in mitochondria and abolishes its toxicity.

scientific article

Impairment of glutamate transport and increased vulnerability to oxidative stress in neuroblastoma SH-SY5Y cells expressing a Cu,Zn superoxide dismutase typical of familial amyotrophic lateral sclerosis.

scientific article published on 8 December 2004

Impairment of mitochondrial calcium handling in a mtSOD1 cell culture model of motoneuron disease

scientific article

Inactivation of cytochrome c oxidase by mutant SOD1s in mouse motoneuronal NSC-34 cells is independent from copper availability but is because of nitric oxide.

scientific article published on 20 October 2009

Inflammatory cytokines increase mitochondrial damage in motoneuronal cells expressing mutant SOD1.

scientific article published on 27 August 2008

Mitochondria and ALS: implications from novel genes and pathways.

scientific article published on 15 June 2012

Mitochondrial dysfunction due to mutant copper/zinc superoxide dismutase associated with amyotrophic lateral sclerosis is reversed by N-acetylcysteine.

scientific article published on August 2003

Mitochondrial redox signalling by p66Shc mediates ALS-like disease through Rac1 inactivation

scientific article published on 09 August 2011

Oxidative inactivation of calcineurin by Cu,Zn superoxide dismutase G93A, a mutant typical of familial amyotrophic lateral sclerosis.

scientific article published in November 2001

Oxidative modulation of nuclear factor-kappaB in human cells expressing mutant fALS-typical superoxide dismutases

scientific article published in December 2002

Phosphatidylinositol 3-kinase is recruited to a specific site in the activated IL-1 receptor I

scholarly article by Sandra Marmiroli published in October 1998

Resistance to striatal dopamine depletion induced by 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine in mice expressing human mutant Cu,Zn superoxide dismutase.

scientific article published in June 2002

Role of mitochondrial raft-like microdomains in the regulation of cell apoptosis.

scientific article published on May 2015

Superoxide dismutase 1 modulates expression of transferrin receptor.

scientific article published on 26 April 2006

The proinflammatory action of microglial P2 receptors is enhanced in SOD1 models for amyotrophic lateral sclerosis.

scientific article

List of works by Alberto Ferri

Acute focal brain damage alters mitochondrial dynamics and autophagy in axotomized neurons.

Apaf1 mediates apoptosis and mitochondrial damage induced by mutant human SOD1s typical of familial amyotrophic lateral sclerosis

Apoptosome inactivation rescues proneural and neural cells from neurodegeneration.

CNF1 Enhances Brain Energy Content and Counteracts Spontaneous Epileptiform Phenomena in Aged DBA/2J Mice

CNF1 increases brain energy level, counteracts neuroinflammatory markers and rescues cognitive deficits in a murine model of Alzheimer's disease

Calcineurin activity is regulated both by redox compounds and by mutant familial amyotrophic lateral sclerosis-superoxide dismutase

Caspase-3 triggers early synaptic dysfunction in a mouse model of Alzheimer's disease

Cell death in amyotrophic lateral sclerosis: interplay between neuronal and glial cells.

Copper depletion increases the mitochondrial-associated SOD1 in neuronal cells

Cysteine 111 affects aggregation and cytotoxicity of mutant Cu,Zn-superoxide dismutase associated with familial amyotrophic lateral sclerosis

Different polymeric forms of actin detected by the fluorescent probe terbium ion ⬇️

Dynamic NAD(P)H post-synaptic autofluorescence signals for the assessment of mitochondrial function in a neurodegenerative disease: monitoring the primary motor cortex of G93A mice, an amyotrophic lateral sclerosis model.

Familial ALS-superoxide dismutases associate with mitochondria and shift their redox potentials.

G-actin modified by plasma membrane interaction polymerizes only in the presence of filamentous myosin

Glutaredoxin 1 is a major player in copper metabolism in neuroblastoma cells.

Glutaredoxin 2 prevents aggregation of mutant SOD1 in mitochondria and abolishes its toxicity.

Impairment of glutamate transport and increased vulnerability to oxidative stress in neuroblastoma SH-SY5Y cells expressing a Cu,Zn superoxide dismutase typical of familial amyotrophic lateral sclerosis.

Impairment of mitochondrial calcium handling in a mtSOD1 cell culture model of motoneuron disease

Inactivation of cytochrome c oxidase by mutant SOD1s in mouse motoneuronal NSC-34 cells is independent from copper availability but is because of nitric oxide.

Inflammatory cytokines increase mitochondrial damage in motoneuronal cells expressing mutant SOD1.

Mitochondria and ALS: implications from novel genes and pathways.

Mitochondrial dysfunction due to mutant copper/zinc superoxide dismutase associated with amyotrophic lateral sclerosis is reversed by N-acetylcysteine.

Mitochondrial redox signalling by p66Shc mediates ALS-like disease through Rac1 inactivation

Oxidative inactivation of calcineurin by Cu,Zn superoxide dismutase G93A, a mutant typical of familial amyotrophic lateral sclerosis.

Oxidative modulation of nuclear factor-kappaB in human cells expressing mutant fALS-typical superoxide dismutases

Phosphatidylinositol 3-kinase is recruited to a specific site in the activated IL-1 receptor I

Resistance to striatal dopamine depletion induced by 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine in mice expressing human mutant Cu,Zn superoxide dismutase.

Role of mitochondrial raft-like microdomains in the regulation of cell apoptosis.

Superoxide dismutase 1 modulates expression of transferrin receptor.

The proinflammatory action of microglial P2 receptors is enhanced in SOD1 models for amyotrophic lateral sclerosis.