List of works - Greg J Barritt

A small component of the endoplasmic reticulum is required for store-operated Ca2+ channel activation in liver cells: evidence from studies using TRPV1 and taurodeoxycholic acid.

scientific article published in March 2009

Arachidonic acid inhibits the store-operated Ca2+ current in rat liver cells.

scientific article

Ca(2+) -permeable channels in the hepatocyte plasma membrane and their roles in hepatocyte physiology.

scientific article published on 07 February 2008

Curcumin inhibits activation of TRPM2 channels in rat hepatocytes

scientific article published on 10 November 2015

Evaluation, using targeted aequorins, of the roles of the endoplasmic reticulum and its (Ca2++Mg2+)ATP-ases in the activation of store-operated Ca2+ channels in liver cells

scientific article published on April 2004

Evidence that Ca2+-release-activated Ca2+ channels in rat hepatocytes are required for the maintenance of hormone-induced Ca2+ oscillations.

scientific article published in March 2003

Evidence that TRPC1 (transient receptor potential canonical 1) forms a Ca2+-permeable channel linked to the regulation of cell volume in liver cells obtained using small interfering RNA targeted against TRPC1 ⬇️

scientific article published on July 15, 2003

Evidence that decreased expression of sinusoidal bile acid transporters accounts for the inhibition by rapamycin of bile flow recovery following liver ischemia

scientific article published on 01 September 2018

Evidence that estrogen receptors play a limited role in mediating enhanced recovery of bile flow in female rats in the acute phase of liver ischemia reperfusion injury

scientific article published on January 1, 2013

Expression and function of TRP channels in liver cells.

scientific article published on January 2011

Expression of Drosophila Ca2+ permeable transient receptor potential-like channel protein in a prostate cancer cell line decreases cell survival.

scientific article published in August 2003

Fast Ca(2+)-dependent inactivation of the store-operated Ca2+ current (ISOC) in liver cells: a role for calmodulin.

scientific article published on 30 April 2004

Glucagon activates Ca2+ and Cl- channels in rat hepatocytes

scientific article published on 31 March 2006

Hepatic ischemia-reperfusion injury: roles of Ca2+ and other intracellular mediators of impaired bile flow and hepatocyte damage.

scientific article published on June 2006

Increased expression of peroxiredoxin 1 and identification of a novel lipid-metabolizing enzyme in the early phase of liver ischemia reperfusion injury.

scientific article

Increasing cycles of intermittent ischemia can effectively maintain liver function during the acute phase of ischemia reperfusion injury by promotion of bile flow and reduction in bile salt toxicity

scientific article published on 01 February 2009

Induction of cholestasis in the perfused rat liver by 2-aminoethyl diphenylborate, an inhibitor of the hepatocyte plasma membrane Ca2+ channels.

scientific article

Inositol trisphosphate analogues selective for types I and II inositol trisphosphate receptors exert differential effects on vasopressin-stimulated Ca2+ inflow and Ca2+ release from intracellular stores in rat hepatocytes.

scientific article

Insulin potentiates EGFR activation and signaling in fibroblasts.

scientific article published on September 2004

Intermittent ischaemia maintains function after ischaemia reperfusion in steatotic livers.

scientific article

Intermittent ischemia but not ischemic preconditioning is effective in restoring bile flow after ischemia reperfusion injury in the livers of aged rats

scientific article published in February 2008

Intermittent ischemia enhances the uptake of indocyanine green to livers subject to ischemia and reperfusion.

scientific article published on 3 May 2012

Ischemic preconditioning and intermittent ischemia preserve bile flow in a rat model of ischemia reperfusion injury

scientific article published on 14 March 2007

Mitochondrial uncoupler FCCP activates proton conductance but does not block store-operated Ca(2+) current in liver cells.

scientific article

Phospholipase C-gamma1 is required for the activation of store-operated Ca2+ channels in liver cells

scientific journal article

Rapamycin induces heme oxygenase-1 in liver but inhibits bile flow recovery after ischemia.

scientific article

Rapamycin induces the expression of heme oxygenase-1 and peroxyredoxin-1 in normal hepatocytes but not in tumorigenic liver cells

scientific article published on 02 October 2018

Regulation of Orai1/STIM1 mediated ICRAC by intracellular pH.

scientific article

Steatosis inhibits liver cell store-operated Ca²⁺ entry and reduces ER Ca²⁺ through a protein kinase C-dependent mechanism

scientific article

Store-operated Ca(2+) channels and Stromal Interaction Molecule 1 (STIM1) are targets for the actions of bile acids on liver cells.

scientific article

Store-operated Ca2+ channels and microdomains of Ca2+ in liver cells.

scientific article published on January 2009

Structural and stoichiometric determinants of Ca2+ release-activated Ca2+ (CRAC) channel Ca2+-dependent inactivation

scientific article published on 26 January 2014

TRPM2 channels mediate acetaminophen-induced liver damage

scientific article published on 03 February 2014

Targeting Ca2+ Signaling in the Initiation, Promotion and Progression of Hepatocellular Carcinoma

scientific article published on 24 September 2020

The glucagon-like peptide-1 analogue exendin-4 reverses impaired intracellular Ca2+ signalling in steatotic hepatocytes

scientific article (publication date: September 2016)

The predominant role of IP₃ type 1 receptors in activation of store-operated Ca²+ entry in liver cells

scientific article published on 20 December 2010

Transient receptor potential and other ion channels as pharmaceutical targets in airway smooth muscle cells.

scientific article

Using continuous porous silicon gradients to study the influence of surface topography on the behaviour of neuroblastoma cells.

scientific article published on November 2007

List of works by Greg J Barritt

A small component of the endoplasmic reticulum is required for store-operated Ca2+ channel activation in liver cells: evidence from studies using TRPV1 and taurodeoxycholic acid.

Arachidonic acid inhibits the store-operated Ca2+ current in rat liver cells.

Ca(2+) -permeable channels in the hepatocyte plasma membrane and their roles in hepatocyte physiology.

Curcumin inhibits activation of TRPM2 channels in rat hepatocytes

Evaluation, using targeted aequorins, of the roles of the endoplasmic reticulum and its (Ca2++Mg2+)ATP-ases in the activation of store-operated Ca2+ channels in liver cells

Evidence that Ca2+-release-activated Ca2+ channels in rat hepatocytes are required for the maintenance of hormone-induced Ca2+ oscillations.

Evidence that TRPC1 (transient receptor potential canonical 1) forms a Ca2+-permeable channel linked to the regulation of cell volume in liver cells obtained using small interfering RNA targeted against TRPC1 ⬇️

Evidence that decreased expression of sinusoidal bile acid transporters accounts for the inhibition by rapamycin of bile flow recovery following liver ischemia

Evidence that estrogen receptors play a limited role in mediating enhanced recovery of bile flow in female rats in the acute phase of liver ischemia reperfusion injury

Expression and function of TRP channels in liver cells.

Expression of Drosophila Ca2+ permeable transient receptor potential-like channel protein in a prostate cancer cell line decreases cell survival.

Fast Ca(2+)-dependent inactivation of the store-operated Ca2+ current (ISOC) in liver cells: a role for calmodulin.

Glucagon activates Ca2+ and Cl- channels in rat hepatocytes

Hepatic ischemia-reperfusion injury: roles of Ca2+ and other intracellular mediators of impaired bile flow and hepatocyte damage.

Increased expression of peroxiredoxin 1 and identification of a novel lipid-metabolizing enzyme in the early phase of liver ischemia reperfusion injury.

Increasing cycles of intermittent ischemia can effectively maintain liver function during the acute phase of ischemia reperfusion injury by promotion of bile flow and reduction in bile salt toxicity

Induction of cholestasis in the perfused rat liver by 2-aminoethyl diphenylborate, an inhibitor of the hepatocyte plasma membrane Ca2+ channels.

Inositol trisphosphate analogues selective for types I and II inositol trisphosphate receptors exert differential effects on vasopressin-stimulated Ca2+ inflow and Ca2+ release from intracellular stores in rat hepatocytes.

Insulin potentiates EGFR activation and signaling in fibroblasts.

Intermittent ischaemia maintains function after ischaemia reperfusion in steatotic livers.

Intermittent ischemia but not ischemic preconditioning is effective in restoring bile flow after ischemia reperfusion injury in the livers of aged rats

Intermittent ischemia enhances the uptake of indocyanine green to livers subject to ischemia and reperfusion.

Ischemic preconditioning and intermittent ischemia preserve bile flow in a rat model of ischemia reperfusion injury

Mitochondrial uncoupler FCCP activates proton conductance but does not block store-operated Ca(2+) current in liver cells.

Phospholipase C-gamma1 is required for the activation of store-operated Ca2+ channels in liver cells

Rapamycin induces heme oxygenase-1 in liver but inhibits bile flow recovery after ischemia.

Rapamycin induces the expression of heme oxygenase-1 and peroxyredoxin-1 in normal hepatocytes but not in tumorigenic liver cells

Regulation of Orai1/STIM1 mediated ICRAC by intracellular pH.

Steatosis inhibits liver cell store-operated Ca²⁺ entry and reduces ER Ca²⁺ through a protein kinase C-dependent mechanism

Store-operated Ca(2+) channels and Stromal Interaction Molecule 1 (STIM1) are targets for the actions of bile acids on liver cells.

Store-operated Ca2+ channels and microdomains of Ca2+ in liver cells.

Structural and stoichiometric determinants of Ca2+ release-activated Ca2+ (CRAC) channel Ca2+-dependent inactivation

TRPM2 channels mediate acetaminophen-induced liver damage

Targeting Ca2+ Signaling in the Initiation, Promotion and Progression of Hepatocellular Carcinoma

The glucagon-like peptide-1 analogue exendin-4 reverses impaired intracellular Ca2+ signalling in steatotic hepatocytes

The predominant role of IP₃ type 1 receptors in activation of store-operated Ca²+ entry in liver cells

Transient receptor potential and other ion channels as pharmaceutical targets in airway smooth muscle cells.

Using continuous porous silicon gradients to study the influence of surface topography on the behaviour of neuroblastoma cells.