List of works - Tali Gidalevitz

A Bystander Mechanism Explains the Specific Phenotype of a Broadly Expressed Misfolded Protein

scientific article

A cellular perspective on conformational disease: the role of genetic background and proteostasis networks

scientific article

Candidate genes that affect aging through protein homeostasis

scientific article published on January 2015

Destabilizing protein polymorphisms in the genetic background direct phenotypic expression of mutant SOD1 toxicity

scientific article (publication date: March 2009)

HSP-4/BiP expression in secretory cells is regulated by a developmental program and not by the unfolded protein response

scientific article published on 25 March 2019

Hsp27 overexpression in the R6/2 mouse model of Huntington's disease: chronic neurodegeneration does not induce Hsp27 activation

scientific article

Identification of the N-terminal peptide binding site of glucose-regulated protein 94.

scientific article

Multiple Molecular Mechanisms Rescue mtDNA Disease in C. elegans

scientific article published in March 2018

Natural genetic variation determines susceptibility to aggregation or toxicity in a C. elegans model for polyglutamine disease

scientific article

Orchestration of secretory protein folding by ER chaperones

scientific article

Progressive disruption of cellular protein folding in models of polyglutamine diseases.

scientific article

Protein disulfide isomerase A6 controls the decay of IRE1α signaling via disulfide-dependent association

scientific article published on 06 February 2014

Quality Control across Compartments-Connecting ERAD with Ribosomal Quality Control

scientific article published on 30 November 2018

Radicicol-sensitive peptide binding to the N-terminal portion of GRP94

scientific article published on 19 August 2002

The peptide-binding activity of GRP94 is regulated by calcium.

scientific article published on July 2007

The stress of protein misfolding: from single cells to multicellular organisms

scientific article

Unexpected cell type-dependent effects of autophagy on polyglutamine aggregation revealed by natural genetic variation in C. elegans

scientific article published on 24 February 2020

List of works by Tali Gidalevitz

A Bystander Mechanism Explains the Specific Phenotype of a Broadly Expressed Misfolded Protein

A cellular perspective on conformational disease: the role of genetic background and proteostasis networks

Candidate genes that affect aging through protein homeostasis

Destabilizing protein polymorphisms in the genetic background direct phenotypic expression of mutant SOD1 toxicity

HSP-4/BiP expression in secretory cells is regulated by a developmental program and not by the unfolded protein response

Hsp27 overexpression in the R6/2 mouse model of Huntington's disease: chronic neurodegeneration does not induce Hsp27 activation

Identification of the N-terminal peptide binding site of glucose-regulated protein 94.

Multiple Molecular Mechanisms Rescue mtDNA Disease in C. elegans

Natural genetic variation determines susceptibility to aggregation or toxicity in a C. elegans model for polyglutamine disease

Orchestration of secretory protein folding by ER chaperones

Progressive disruption of cellular protein folding in models of polyglutamine diseases.

Protein disulfide isomerase A6 controls the decay of IRE1α signaling via disulfide-dependent association

Quality Control across Compartments-Connecting ERAD with Ribosomal Quality Control

Radicicol-sensitive peptide binding to the N-terminal portion of GRP94

The peptide-binding activity of GRP94 is regulated by calcium.

The stress of protein misfolding: from single cells to multicellular organisms

Unexpected cell type-dependent effects of autophagy on polyglutamine aggregation revealed by natural genetic variation in C. elegans