List of works - Gretchen V Gee

Abstract 3001: MicroRNAs regulating wnt signaling are downregulated in malignant pleural mesothelioma

Derivation of a JC virus-resistant human glial cell line: implications for the identification of host cell factors that determine viral tropism

scientific article published on September 2003

Downregulated microRNAs in the differential diagnosis of malignant pleural mesothelioma

journal article published in 2010

Host cell autophagy promotes BK virus infection

scientific article published on 2 April 2014

Interaction between Simian Virus 40 Major Capsid Protein VP1 and Cell Surface Ganglioside GM1 Triggers Vacuole Formation

scientific article

JC Virus infected choroid plexus epithelial cells produce extracellular vesicles that infect glial cells independently of the virus attachment receptor

scientific article published on 04 March 2020

Microarray analysis of glial cells resistant to JCV infection suggests a correlation between viral infection and inflammatory cytokine gene expression

scientific article

Modeling a sialic acid binding pocket in the external loops of JC virus VP1.

scientific article published on 3 September 2004

Modulation of a pore in the capsid of JC polyomavirus reduces infectivity and prevents exposure of the minor capsid proteins.

scientific article

NFAT4 is required for JC virus infection of glial cells

scientific article

Progressive Multifocal Leukoencephalopathy-Associated Mutations in the JC Polyomavirus Capsid Disrupt Lactoseries Tetrasaccharide c Binding

scientific article

Pseudovirus mimics cell entry and trafficking of the human polyomavirus JCPyV.

scientific article published on 04 October 2013

Role of N-linked glycosylation of the 5-HT2A receptor in JC virus infection.

scientific article

The Greater Affinity of JC Polyomavirus Capsid for α2,6-Linked Lactoseries Tetrasaccharide c than for Other Sialylated Glycans Is a Major Determinant of Infectivity

scientific article published on 8 April 2015

The human polyomavirus, JCV, uses serotonin receptors to infect cells

scientific article

The role of sialic acid in human polyomavirus infections

scientific article

Trichodysplasia spinulosa-Associated Polyomavirus Uses a Displaced Binding Site on VP1 to Engage Sialylated Glycolipids

scientific article published on 24 August 2015

List of works by Gretchen V Gee

Abstract 3001: MicroRNAs regulating wnt signaling are downregulated in malignant pleural mesothelioma

Derivation of a JC virus-resistant human glial cell line: implications for the identification of host cell factors that determine viral tropism

Downregulated microRNAs in the differential diagnosis of malignant pleural mesothelioma

Host cell autophagy promotes BK virus infection

Interaction between Simian Virus 40 Major Capsid Protein VP1 and Cell Surface Ganglioside GM1 Triggers Vacuole Formation

JC Virus infected choroid plexus epithelial cells produce extracellular vesicles that infect glial cells independently of the virus attachment receptor

Microarray analysis of glial cells resistant to JCV infection suggests a correlation between viral infection and inflammatory cytokine gene expression

Modeling a sialic acid binding pocket in the external loops of JC virus VP1.

Modulation of a pore in the capsid of JC polyomavirus reduces infectivity and prevents exposure of the minor capsid proteins.

NFAT4 is required for JC virus infection of glial cells

Progressive Multifocal Leukoencephalopathy-Associated Mutations in the JC Polyomavirus Capsid Disrupt Lactoseries Tetrasaccharide c Binding

Pseudovirus mimics cell entry and trafficking of the human polyomavirus JCPyV.

Role of N-linked glycosylation of the 5-HT2A receptor in JC virus infection.

The Greater Affinity of JC Polyomavirus Capsid for α2,6-Linked Lactoseries Tetrasaccharide c than for Other Sialylated Glycans Is a Major Determinant of Infectivity

The human polyomavirus, JCV, uses serotonin receptors to infect cells

The role of sialic acid in human polyomavirus infections

Trichodysplasia spinulosa-Associated Polyomavirus Uses a Displaced Binding Site on VP1 to Engage Sialylated Glycolipids