List of works - Catherine A Blizzard

A Case for Microtubule Vulnerability in Amyotrophic Lateral Sclerosis: Altered Dynamics During Disease

scientific article

Amyotrophic lateral sclerosis associated mislocalisation of TDP-43 to the cytoplasm causes cortical hyperexcitability and reduced excitatory neurotransmission in the motor cortex

scientific article published on 16 October 2020

Amyotrophic lateral sclerosis mutant TDP-43 may cause synaptic dysfunction through altered dendritic spine function

scientific article published on 17 May 2019

Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

scientific article published on 30 March 2016

Axonal shearing in mature cortical neurons induces attempted regeneration and the reestablishment of neurite polarity

scientific article published on 26 August 2009

Axonopathy and cytoskeletal disruption in degenerative diseases of the central nervous system.

scientific article published on 13 August 2009

Calretinin and Neuropeptide Y interneurons are differentially altered in the motor cortex of the SOD1G93A mouse model of ALS.

scientific article published on 15 March 2017

Chronic excitotoxin-induced axon degeneration in a compartmented neuronal culture model

scientific article published on 23 February 2012

Cortical murine neurons lacking the neurofilament light chain protein have an attenuated response to injury in vitro.

scientific article published on November 2013

Degeneration of axons in spinal white matter in G93A mSOD1 mouse characterized by NFL and α-internexin immunoreactivity

scientific article published on 17 May 2012

Efficacy of epothilones in central nervous system trauma treatment: what has age got to do with it?

scientific article published on 01 April 2021

Epothilone D accelerates disease progression in the SOD1G93A mouse model of Amyotrophic Lateral Sclerosis

scientific article published on 30 January 2018

Excitotoxicity mediated by non-NMDA receptors causes distal axonopathy in long-term cultured spinal motor neurons.

scientific article published on October 2007

Focal damage to the adult rat neocortex induces wound healing accompanied by axonal sprouting and dendritic structural plasticity

scientific article published on 28 May 2010

Identifying the primary site of pathogenesis in amyotrophic lateral sclerosis - vulnerability of lower motor neurons to proximal excitotoxicity

scientific article

Inducing Chronic Excitotoxicity in the Mouse Spinal Cord to Investigate Lower Motor Neuron Degeneration

scientific article published on 2 March 2016

Inhibitory dysfunction in amyotrophic lateral sclerosis: future therapeutic opportunities

scientific article

Microfluidic primary culture model of the lower motor neuron-neuromuscular junction circuit

scientific article published on 14 June 2013

Mild Traumatic Brain Injury Leads to Decreased Inhibition and a Differential Response of Calretinin Positive Interneurons in the Injured Cortex.

scientific article published on 2 May 2017

Mild and repetitive very mild axonal stretch injury triggers cystoskeletal mislocalization and growth cone collapse

scientific article

Reduced Excitability and Increased Neurite Complexity of Cortical Interneurons in a Familial Mouse Model of Amyotrophic Lateral Sclerosis

scientific article published on 28 September 2018

Synapse Dysfunction of Layer V Pyramidal Neurons Precedes Neurodegeneration in a Mouse Model of TDP-43 Proteinopathies.

scientific article published on 5 August 2016

The Microtubule-Modulating Drug Epothilone D Alters Dendritic Spine Morphology in a Mouse Model of Mild Traumatic Brain Injury

scientific article published on 30 July 2018

The Tasmanian devil transcriptome reveals Schwann cell origins of a clonally transmissible cancer

scientific article

The degree of astrocyte activation in multiple system atrophy is inversely proportional to the distance to α-synuclein inclusions

scientific article published on 27 February 2015

The microtubule-stabilizing drug Epothilone D increases axonal sprouting following transection injury in vitro.

scientific article published on 12 February 2015

The pathologic outcomes and efficacy of epothilone treatment following traumatic brain injury is determined by age

scientific article published on 08 April 2020

List of works by Catherine A Blizzard

A Case for Microtubule Vulnerability in Amyotrophic Lateral Sclerosis: Altered Dynamics During Disease

Amyotrophic lateral sclerosis associated mislocalisation of TDP-43 to the cytoplasm causes cortical hyperexcitability and reduced excitatory neurotransmission in the motor cortex

Amyotrophic lateral sclerosis mutant TDP-43 may cause synaptic dysfunction through altered dendritic spine function

Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

Axonal shearing in mature cortical neurons induces attempted regeneration and the reestablishment of neurite polarity

Axonopathy and cytoskeletal disruption in degenerative diseases of the central nervous system.

Calretinin and Neuropeptide Y interneurons are differentially altered in the motor cortex of the SOD1G93A mouse model of ALS.

Chronic excitotoxin-induced axon degeneration in a compartmented neuronal culture model

Cortical murine neurons lacking the neurofilament light chain protein have an attenuated response to injury in vitro.

Degeneration of axons in spinal white matter in G93A mSOD1 mouse characterized by NFL and α-internexin immunoreactivity

Efficacy of epothilones in central nervous system trauma treatment: what has age got to do with it?

Epothilone D accelerates disease progression in the SOD1G93A mouse model of Amyotrophic Lateral Sclerosis

Excitotoxicity mediated by non-NMDA receptors causes distal axonopathy in long-term cultured spinal motor neurons.

Focal damage to the adult rat neocortex induces wound healing accompanied by axonal sprouting and dendritic structural plasticity

Identifying the primary site of pathogenesis in amyotrophic lateral sclerosis - vulnerability of lower motor neurons to proximal excitotoxicity

Inducing Chronic Excitotoxicity in the Mouse Spinal Cord to Investigate Lower Motor Neuron Degeneration

Inhibitory dysfunction in amyotrophic lateral sclerosis: future therapeutic opportunities

Microfluidic primary culture model of the lower motor neuron-neuromuscular junction circuit

Mild Traumatic Brain Injury Leads to Decreased Inhibition and a Differential Response of Calretinin Positive Interneurons in the Injured Cortex.

Mild and repetitive very mild axonal stretch injury triggers cystoskeletal mislocalization and growth cone collapse

Reduced Excitability and Increased Neurite Complexity of Cortical Interneurons in a Familial Mouse Model of Amyotrophic Lateral Sclerosis

Synapse Dysfunction of Layer V Pyramidal Neurons Precedes Neurodegeneration in a Mouse Model of TDP-43 Proteinopathies.

The Microtubule-Modulating Drug Epothilone D Alters Dendritic Spine Morphology in a Mouse Model of Mild Traumatic Brain Injury

The Tasmanian devil transcriptome reveals Schwann cell origins of a clonally transmissible cancer

The degree of astrocyte activation in multiple system atrophy is inversely proportional to the distance to α-synuclein inclusions

The microtubule-stabilizing drug Epothilone D increases axonal sprouting following transection injury in vitro.

The pathologic outcomes and efficacy of epothilone treatment following traumatic brain injury is determined by age