List of works - Vladimir L. Gabai

Adaptation of Ehrlich ascites carcinoma cells to energy deprivation in vivo can be associated with heat shock protein accumulation.

scientific article published in October 1995

Anticancer Effects of Targeting Hsp70 in Tumor Stromal Cells.

scientific article published on 8 August 2016

Association of blebbing with assembly of cytoskeletal proteins in ATP-depleted EL-4 ascites tumour cells.

scientific article published in January 1992

Change in energy metabolism of ascites cancer cells with a decrease in pH

scientific article published on September 1, 1991

Determination of cell survival or death.

scientific article

Disruption of plasma cell membrane permeability for Ca2+ ions in radiation-induced apoptosis of thymocytes

scientific article published on March 1, 1997

Distinct effects of heat shock and ATP depletion on distribution and isoform patterns of human Hsp27 in endothelial cells

scientific article published on 01 August 1996

Distinct hsp70 domains mediate apoptosis-inducing factor release and nuclear accumulation

scientific article published on 4 January 2006

Effect of protein inhibitors of mitochondrial ATPase in intact rat thymocytes and Ehrlich ascites carcinoma cells

scientific article published on July 1, 1995

Effective expression of recombinant cytotoxic protein via its attachment to a polyglutamine domain.

scientific article

Feasibility analysis of p62 (SQSTM1)-encoding DNA vaccine as a novel cancer immunotherapy.

scientific article published on October 2014

Glucose decreases respiratory control ratio in EL-4 tumor cells

scientific article published on November 23, 1992

HSP72 depletion suppresses gammaH2AX activation by genotoxic stresses via p53/p21 signaling

scientific article published on 11 January 2010

Heat shock protein Hsp72 controls oncogene-induced senescence pathways in cancer cells.

scientific article

Heat shock protein Hsp72 plays an essential role in Her2-induced mammary tumorigenesis ⬇️

scientific article published on February 7, 2011

Heat shock transcription factor Hsf1 is involved in tumor progression via regulation of hypoxia-inducible factor 1 and RNA-binding protein HuR

scientific article published on 3 January 2012

High levels of heat shock protein Hsp72 in cancer cells suppress default senescence pathways

scientific article published on 01 March 2007

Hsp70 in cancer: back to the future

scientific article

Hsp70 prevents activation of stress kinases. A novel pathway of cellular thermotolerance

scientific article published on 01 July 1997

Hsp70-Bag3 interactions regulate cancer-related signaling networks

scientific article published on 3 July 2014

Hsp72 and stress kinase c-jun N-terminal kinase regulate the bid-dependent pathway in tumor necrosis factor-induced apoptosis

scientific article

Hsp90 inhibitors as promising agents for radiotherapy

scientific article

Inactivation of Dual-Specificity Phosphatases Is Involved in the Regulation of Extracellular Signal-Regulated Kinases by Heat Shock and Hsp72 ⬇️

scientific article published on June 1, 2003

Increased expression of the major heat shock protein Hsp72 in human prostate carcinoma cells is dispensable for their viability but confers resistance to a variety of anticancer agents.

scientific article published on May 2005

Induction of heat-shock protein synthesis and thermotolerance in EL-4 ascites tumor cells by transient ATP depletion after ischemic stress.

scientific article published in April 1994

Inhibition of influenza M2-induced cell death alleviates its negative contribution to vaccination efficiency

scientific article

Inhibition of uncoupled respiration in tumor cells. A possible role of mitochondrial Ca2+ efflux

scientific article published on 01 August 1993

Intracellular aggregation of polypeptides with expanded polyglutamine domain is stimulated by stress-activated kinase MEKK1.

scientific article

Lack of serum causes apoptosis of thymocytes not requiring protein synthesis and ATP generation

scientific article published on 01 August 1995

Mechanisms of tumor cell necrosis.

scientific article published on January 2010

Molecular chaperones regulate p53 and suppress senescence programs.

scientific article published on 25 May 2007

Multiple thermometers in mammalian cells: why do cells from homeothermic organisms need to measure temperature?

scientific article

Natural hidden antibodies reacting with DNA or cardiolipin bind to thymocytes and evoke their death.

scientific article published in August 1997

Necrosis is an active and controlled form of programmed cell death.

scientific article

Oncogenes induce senescence with incomplete growth arrest and suppress the DNA damage response in immortalized cells.

scientific article

Prime-boost vaccination with a combination of proteosome-degradable and wild-type forms of two influenza proteins leads to augmented CTL response.

scientific article published on 14 March 2008

Proteasome failure promotes positioning of lysosomes around the aggresome via local block of microtubule-dependent transport

scientific article

Proteasome inhibitors activate stress kinases and induce Hsp72. Diverse effects on apoptosis

scientific article published on 01 March 1998

Protein aggregation as primary and characteristic cell reaction to various stresses

scientific article published on 01 August 1993

Proteotoxicity is not the reason for the dependence of cancer cells on the major chaperone Hsp70

scientific article

Regulation of necrosis of H9c2 myogenic cells upon transient energy deprivation. Rapid deenergization of mitochondria precedes necrosis and is controlled by reactive oxygen species, stress kinase JNK, HSP72 and ARC.

scientific article published on 30 September 2003

Resistance of Ehrlich tumor cells to apoptosis can be due to accumulation of heat shock proteins.

scientific article published in November 1995

Rise in heat-shock protein level confers tolerance to energy deprivation

scientific article published on 01 August 1993

Targeting heat shock response to sensitize cancer cells to proteasome and Hsp90 inhibitors

scientific article published in February 2006

The function of HSP72 in suppression of c-Jun N-terminal kinase activation can be dissociated from its role in prevention of protein damage

scientific article published on 01 July 1999

The heat shock transcription factor Hsf1 is downregulated in DNA damage-associated senescence, contributing to the maintenance of senescence phenotype.

scientific article

Triggering aggresome formation. Dissecting aggresome-targeting and aggregation signals in synphilin 1

scientific article

Tumor cell resistance to energy deprivation and hyperthermia can be determined by the actin skeleton stability.

scientific article published in June 1993

List of works by Vladimir L. Gabai

Adaptation of Ehrlich ascites carcinoma cells to energy deprivation in vivo can be associated with heat shock protein accumulation.

Anticancer Effects of Targeting Hsp70 in Tumor Stromal Cells.

Association of blebbing with assembly of cytoskeletal proteins in ATP-depleted EL-4 ascites tumour cells.

Change in energy metabolism of ascites cancer cells with a decrease in pH

Determination of cell survival or death.

Disruption of plasma cell membrane permeability for Ca2+ ions in radiation-induced apoptosis of thymocytes

Distinct effects of heat shock and ATP depletion on distribution and isoform patterns of human Hsp27 in endothelial cells

Distinct hsp70 domains mediate apoptosis-inducing factor release and nuclear accumulation

Effect of protein inhibitors of mitochondrial ATPase in intact rat thymocytes and Ehrlich ascites carcinoma cells

Effective expression of recombinant cytotoxic protein via its attachment to a polyglutamine domain.

Feasibility analysis of p62 (SQSTM1)-encoding DNA vaccine as a novel cancer immunotherapy.

Glucose decreases respiratory control ratio in EL-4 tumor cells

HSP72 depletion suppresses gammaH2AX activation by genotoxic stresses via p53/p21 signaling

Heat shock protein Hsp72 controls oncogene-induced senescence pathways in cancer cells.

Heat shock protein Hsp72 plays an essential role in Her2-induced mammary tumorigenesis ⬇️

Heat shock transcription factor Hsf1 is involved in tumor progression via regulation of hypoxia-inducible factor 1 and RNA-binding protein HuR

High levels of heat shock protein Hsp72 in cancer cells suppress default senescence pathways

Hsp70 in cancer: back to the future

Hsp70 prevents activation of stress kinases. A novel pathway of cellular thermotolerance

Hsp70-Bag3 interactions regulate cancer-related signaling networks

Hsp72 and stress kinase c-jun N-terminal kinase regulate the bid-dependent pathway in tumor necrosis factor-induced apoptosis

Hsp90 inhibitors as promising agents for radiotherapy

Inactivation of Dual-Specificity Phosphatases Is Involved in the Regulation of Extracellular Signal-Regulated Kinases by Heat Shock and Hsp72 ⬇️

Increased expression of the major heat shock protein Hsp72 in human prostate carcinoma cells is dispensable for their viability but confers resistance to a variety of anticancer agents.

Induction of heat-shock protein synthesis and thermotolerance in EL-4 ascites tumor cells by transient ATP depletion after ischemic stress.

Inhibition of influenza M2-induced cell death alleviates its negative contribution to vaccination efficiency

Inhibition of uncoupled respiration in tumor cells. A possible role of mitochondrial Ca2+ efflux

Intracellular aggregation of polypeptides with expanded polyglutamine domain is stimulated by stress-activated kinase MEKK1.

Lack of serum causes apoptosis of thymocytes not requiring protein synthesis and ATP generation

Mechanisms of tumor cell necrosis.

Molecular chaperones regulate p53 and suppress senescence programs.

Multiple thermometers in mammalian cells: why do cells from homeothermic organisms need to measure temperature?

Natural hidden antibodies reacting with DNA or cardiolipin bind to thymocytes and evoke their death.

Necrosis is an active and controlled form of programmed cell death.

Oncogenes induce senescence with incomplete growth arrest and suppress the DNA damage response in immortalized cells.

Prime-boost vaccination with a combination of proteosome-degradable and wild-type forms of two influenza proteins leads to augmented CTL response.

Proteasome failure promotes positioning of lysosomes around the aggresome via local block of microtubule-dependent transport

Proteasome inhibitors activate stress kinases and induce Hsp72. Diverse effects on apoptosis

Protein aggregation as primary and characteristic cell reaction to various stresses

Proteotoxicity is not the reason for the dependence of cancer cells on the major chaperone Hsp70

Regulation of necrosis of H9c2 myogenic cells upon transient energy deprivation. Rapid deenergization of mitochondria precedes necrosis and is controlled by reactive oxygen species, stress kinase JNK, HSP72 and ARC.

Resistance of Ehrlich tumor cells to apoptosis can be due to accumulation of heat shock proteins.

Rise in heat-shock protein level confers tolerance to energy deprivation

Targeting heat shock response to sensitize cancer cells to proteasome and Hsp90 inhibitors

The function of HSP72 in suppression of c-Jun N-terminal kinase activation can be dissociated from its role in prevention of protein damage

The heat shock transcription factor Hsf1 is downregulated in DNA damage-associated senescence, contributing to the maintenance of senescence phenotype.

Triggering aggresome formation. Dissecting aggresome-targeting and aggregation signals in synphilin 1

Tumor cell resistance to energy deprivation and hyperthermia can be determined by the actin skeleton stability.