List of works - Robert Maile

Antigen-Specific Modulation of an Immune Response by In Vivo Administration of Soluble MHC Class I Tetramers

article

Association between early airway damage-associated molecular patterns and subsequent bacterial infection in patients with inhalational and burn injury

scientific article published on 13 March 2015

Blocking CXCL1-dependent neutrophil recruitment prevents immune damage and reduces pulmonary bacterial infection after inhalation injury.

scientific article

CD8(+) T cells express a T-helper 1--like phenotype after burn injury

scientific article published on 01 August 2001

CD8+ T cell activation is governed by TCR-peptide/MHC affinity, not dissociation rate

scientific article published on 01 September 2007

Direct detection of blood nitric oxide reveals a burn-dependent decrease of nitric oxide in response to Pseudomonas aeruginosa infection.

scientific article published on 4 June 2016

Flagellin treatment prevents increased susceptibility to systemic bacterial infection after injury by inhibiting anti-inflammatory IL-10+ IL-12- neutrophil polarization

scientific article published on 15 January 2014

Increased Toll-like receptor 4 expression on T cells may be a mechanism for enhanced T cell response late after burn injury.

scientific article published in August 2006

Innate Immune Cell Recovery Is Positively Regulated by NLRP12 during Emergency Hematopoiesis

scientific article

Interplay between TCR affinity and necessity of coreceptor ligation: high-affinity peptide-MHC/TCR interaction overcomes lack of CD8 engagement

scientific journal article

Low-avidity CD8lo T cells induced by incomplete antigen stimulation in vivo regulate naive higher avidity CD8hi T cell responses to the same antigen.

scientific article published in February 2006

Lymphopenia-induced homeostatic proliferation of CD8+ T cells is a mechanism for effective allogeneic skin graft rejection following burn injury.

scientific article published in June 2006

Memory CD8 T cells require CD8 coreceptor engagement for calcium mobilization and proliferation, but not cytokine production.

scientific article published in January 2005

Naive CD8+ T cells do not require costimulation for proliferation and differentiation into cytotoxic effector cells.

scientific article published in February 2000

One-hit wonder: Late after burn injury, granulocytes can clear one bacterial infection but cannot control a subsequent infection

scientific article published on 02 March 2019

Peptidic termini play a significant role in TCR recognition

scientific article

Peripheral "CD8 tuning" dynamically modulates the size and responsiveness of an antigen-specific T cell pool in vivo.

scientific article published in January 2005

Pseudomonas aeruginosa exoproducts determine antibiotic efficacy against Staphylococcus aureus.

scientific article published on 27 November 2017

The effect of burn injury on CD8+ and CD4+ T cells in an irradiation model of homeostatic proliferation.

scientific article published in November 2006

Timeline of health care-associated infections and pathogens after burn injuries

scientific article

Transgene expression levels and kinetics determine risk of humoral immune response modeled in factor IX knockout and missense mutant mice.

scientific article published on 26 October 2006

List of works by Robert Maile

Antigen-Specific Modulation of an Immune Response by In Vivo Administration of Soluble MHC Class I Tetramers

Association between early airway damage-associated molecular patterns and subsequent bacterial infection in patients with inhalational and burn injury

Blocking CXCL1-dependent neutrophil recruitment prevents immune damage and reduces pulmonary bacterial infection after inhalation injury.

CD8(+) T cells express a T-helper 1--like phenotype after burn injury

CD8+ T cell activation is governed by TCR-peptide/MHC affinity, not dissociation rate

Direct detection of blood nitric oxide reveals a burn-dependent decrease of nitric oxide in response to Pseudomonas aeruginosa infection.

Flagellin treatment prevents increased susceptibility to systemic bacterial infection after injury by inhibiting anti-inflammatory IL-10+ IL-12- neutrophil polarization

Increased Toll-like receptor 4 expression on T cells may be a mechanism for enhanced T cell response late after burn injury.

Innate Immune Cell Recovery Is Positively Regulated by NLRP12 during Emergency Hematopoiesis

Interplay between TCR affinity and necessity of coreceptor ligation: high-affinity peptide-MHC/TCR interaction overcomes lack of CD8 engagement

Low-avidity CD8lo T cells induced by incomplete antigen stimulation in vivo regulate naive higher avidity CD8hi T cell responses to the same antigen.

Lymphopenia-induced homeostatic proliferation of CD8+ T cells is a mechanism for effective allogeneic skin graft rejection following burn injury.

Memory CD8 T cells require CD8 coreceptor engagement for calcium mobilization and proliferation, but not cytokine production.

Naive CD8+ T cells do not require costimulation for proliferation and differentiation into cytotoxic effector cells.

One-hit wonder: Late after burn injury, granulocytes can clear one bacterial infection but cannot control a subsequent infection

Peptidic termini play a significant role in TCR recognition

Peripheral "CD8 tuning" dynamically modulates the size and responsiveness of an antigen-specific T cell pool in vivo.

Pseudomonas aeruginosa exoproducts determine antibiotic efficacy against Staphylococcus aureus.

The effect of burn injury on CD8+ and CD4+ T cells in an irradiation model of homeostatic proliferation.

Timeline of health care-associated infections and pathogens after burn injuries

Transgene expression levels and kinetics determine risk of humoral immune response modeled in factor IX knockout and missense mutant mice.