List of works - Yasushi Kishimoto

A role for myosin Va in cerebellar plasticity and motor learning: a possible mechanism underlying neurological disorder in myosin Va disease

scientific article

Age-dependent impairment of delay and trace eyeblink conditioning in mice

scientific article published in October 2001

Age-dependent impairment of eyeblink conditioning in prion protein-deficient mice.

scientific article

Altered Cortical Dynamics and Cognitive Function upon Haploinsufficiency of the Autism-Linked Excitatory Synaptic Suppressor MDGA2.

scientific article published in September 2016

Classical eyeblink conditioning in glutamate receptor subunit delta 2 mutant mice is impaired in the delay paradigm but not in the trace paradigm.

scientific article published in March 2001

Conditioned eyeblink learning is formed and stored without cerebellar granule cell transmission

scholarly article

Conditioned eyeblink response is impaired in mutant mice lacking NMDA receptor subunit NR2A ⬇️

scientific article published on December 1, 1997

Data on amyloid precursor protein accumulation, spontaneous physical activity, and motor learning after traumatic brain injury in the triple-transgenic mouse model of Alzheimer׳s disease

scientific article

Early Contextual Fear Memory Deficits in a Double-Transgenic Amyloid-β Precursor Protein/Presenilin 2 Mouse Model of Alzheimer's Disease

scientific article published on 27 November 2017

Early impairment in a water-finding test in a longitudinal study of the Tg2576 mouse model of Alzheimer's disease.

scientific article

Effects of the noncompetitive NMDA receptor antagonist MK-801 on classical eyeblink conditioning in mice.

scientific article

Endogenous cannabinoid signaling through the CB1 receptor is essential for cerebellum-dependent discrete motor learning

scientific article

Hippocampal CA3 NMDA receptors are crucial for adaptive timing of trace eyeblink conditioned response.

scientific article published in February 2006

Impaired delay but normal trace eyeblink conditioning in PLCbeta4 mutant mice

scientific article published in September 2001

Impaired long-trace eyeblink conditioning in a Tg2576 mouse model of Alzheimer's disease

scientific article published on 6 November 2011

Impairment of eyeblink conditioning in GluRdelta2-mutant mice depends on the temporal overlap between conditioned and unconditioned stimuli.

scientific article published in November 2001

Implicit Memory in Monkeys: Development of a Delay Eyeblink Conditioning System with Parallel Electromyographic and High-Speed Video Measurements

scientific article

Inhibition of MAO‐A and stimulation of behavioural activities in mice by the inactive prodrug form of the anti‐influenza agent oseltamivir ⬇️

scientific article published on May 1, 2013

Involvement of protein-tyrosine phosphatase PTPMEG in motor learning and cerebellar long-term depression

scientific article published in October 2007

Junctophilin-mediated channel crosstalk essential for cerebellar synaptic plasticity.

scientific article

Loss of Synapse Repressor MDGA1 Enhances Perisomatic Inhibition, Confers Resistance to Network Excitation, and Impairs Cognitive Function

scientific article published in December 2017

Phospholipase Cbeta4 and protein kinase Calpha and/or protein kinase CbetaI are involved in the induction of long term depression in cerebellar Purkinje cells.

scientific article published on 10 September 2001

Presenilin 2 mutation accelerates the onset of impairment in trace eyeblink conditioning in a mouse model of Alzheimer's disease overexpressing human mutant amyloid precursor protein.

scientific article published on 28 January 2013

Task-specific enhancement of hippocampus-dependent learning in mice deficient in monoacylglycerol lipase, the major hydrolyzing enzyme of the endocannabinoid 2-arachidonoylglycerol

scientific article published on 2 June 2015

The synaptic targeting of mGluR1 by its carboxyl-terminal domain is crucial for cerebellar function.

scientific article published in February 2014

Traumatic Brain Injury by Weight-Drop Method Causes Transient Amyloid- Deposition and Acute Cognitive Deficits in Mice

scientific article published on 03 March 2019

Traumatic brain injury accelerates amyloid-β deposition and impairs spatial learning in the triple-transgenic mouse model of Alzheimer's disease

scientific article published on 29 June 2016

mGluR1 in cerebellar Purkinje cells is essential for the formation but not expression of associative eyeblink memory

scientific article published on 14 May 2019

mGluR1 in cerebellar Purkinje cells is required for normal association of temporally contiguous stimuli in classical conditioning.

scientific article

List of works by Yasushi Kishimoto

A role for myosin Va in cerebellar plasticity and motor learning: a possible mechanism underlying neurological disorder in myosin Va disease

Age-dependent impairment of delay and trace eyeblink conditioning in mice

Age-dependent impairment of eyeblink conditioning in prion protein-deficient mice.

Altered Cortical Dynamics and Cognitive Function upon Haploinsufficiency of the Autism-Linked Excitatory Synaptic Suppressor MDGA2.

Classical eyeblink conditioning in glutamate receptor subunit delta 2 mutant mice is impaired in the delay paradigm but not in the trace paradigm.

Conditioned eyeblink learning is formed and stored without cerebellar granule cell transmission

Conditioned eyeblink response is impaired in mutant mice lacking NMDA receptor subunit NR2A ⬇️

Data on amyloid precursor protein accumulation, spontaneous physical activity, and motor learning after traumatic brain injury in the triple-transgenic mouse model of Alzheimer׳s disease

Early Contextual Fear Memory Deficits in a Double-Transgenic Amyloid-β Precursor Protein/Presenilin 2 Mouse Model of Alzheimer's Disease

Early impairment in a water-finding test in a longitudinal study of the Tg2576 mouse model of Alzheimer's disease.

Effects of the noncompetitive NMDA receptor antagonist MK-801 on classical eyeblink conditioning in mice.

Endogenous cannabinoid signaling through the CB1 receptor is essential for cerebellum-dependent discrete motor learning

Hippocampal CA3 NMDA receptors are crucial for adaptive timing of trace eyeblink conditioned response.

Impaired delay but normal trace eyeblink conditioning in PLCbeta4 mutant mice

Impaired long-trace eyeblink conditioning in a Tg2576 mouse model of Alzheimer's disease

Impairment of eyeblink conditioning in GluRdelta2-mutant mice depends on the temporal overlap between conditioned and unconditioned stimuli.

Implicit Memory in Monkeys: Development of a Delay Eyeblink Conditioning System with Parallel Electromyographic and High-Speed Video Measurements

Inhibition of MAO‐A and stimulation of behavioural activities in mice by the inactive prodrug form of the anti‐influenza agent oseltamivir ⬇️

Involvement of protein-tyrosine phosphatase PTPMEG in motor learning and cerebellar long-term depression

Junctophilin-mediated channel crosstalk essential for cerebellar synaptic plasticity.

Loss of Synapse Repressor MDGA1 Enhances Perisomatic Inhibition, Confers Resistance to Network Excitation, and Impairs Cognitive Function

Phospholipase Cbeta4 and protein kinase Calpha and/or protein kinase CbetaI are involved in the induction of long term depression in cerebellar Purkinje cells.

Presenilin 2 mutation accelerates the onset of impairment in trace eyeblink conditioning in a mouse model of Alzheimer's disease overexpressing human mutant amyloid precursor protein.

Task-specific enhancement of hippocampus-dependent learning in mice deficient in monoacylglycerol lipase, the major hydrolyzing enzyme of the endocannabinoid 2-arachidonoylglycerol

The synaptic targeting of mGluR1 by its carboxyl-terminal domain is crucial for cerebellar function.

Traumatic Brain Injury by Weight-Drop Method Causes Transient Amyloid- Deposition and Acute Cognitive Deficits in Mice

Traumatic brain injury accelerates amyloid-β deposition and impairs spatial learning in the triple-transgenic mouse model of Alzheimer's disease

mGluR1 in cerebellar Purkinje cells is essential for the formation but not expression of associative eyeblink memory

mGluR1 in cerebellar Purkinje cells is required for normal association of temporally contiguous stimuli in classical conditioning.