List of works - Lin Kooi Ong

Authors' response re: "Reconsidering the role of glial cells in chronic stress-induced dopaminergic neurons loss within the substantia nigra? Friend of foe?" by Ong et al. Brain Behavior and Immunity, 2016.

scientific article published on 30 November 2016

Chronic stress exposure following photothrombotic stroke is associated with increased levels of Amyloid beta accumulation and altered oligomerisation at sites of thalamic secondary neurodegeneration in mice

scientific article published on 24 June 2016

Chronic stress induced disruption of the peri-infarct neurovascular unit following experimentally induced photothrombotic stroke.

scientific article

Early life peripheral lipopolysaccharide challenge reprograms catecholaminergic neurons

scientific article published on 10 January 2017

Early life stress and post-weaning high fat diet alter tyrosine hydroxylase regulation and AT1 receptor expression in the adrenal gland in a sex dependent manner.

scientific article published on 7 February 2013

Functional programming of the autonomic nervous system by early life immune exposure: implications for anxiety.

scientific article

Growth Hormone Deficiency Is Frequent After Recent Stroke

article

Growth Hormone Promotes Motor Function after Experimental Stroke and Enhances Recovery-Promoting Mechanisms within the Peri-Infarct Area

scientific article published on 17 January 2020

Growth Hormone Treatment Promotes Remote Hippocampal Plasticity after Experimental Cortical Stroke

scientific article published on 26 June 2020

Impaired microglia process dynamics post-stroke are specific to sites of secondary neurodegeneration.

scientific article

Increased microglial activation in the rat brain following neonatal exposure to a bacterial mimetic

scientific article published in September 2011

Neonatal overfeeding increases capacity for catecholamine biosynthesis from the adrenal gland acutely and long-term in the male rat.

scientific article published on 25 November 2017

Neurobiological consequences of acute footshock stress: effects on tyrosine hydroxylase phosphorylation and activation in the rat brain and adrenal medulla.

scientific article published on 30 October 2013

Neurological impairment caused by Schistosoma mansoni systemic infection exhibits early features of idiopathic neurodegenerative disease

scientific article published on 22 July 2021

Oral administration of corticosterone at stress-like levels drives microglial but not vascular disturbances post-stroke

scientific article published on 10 March 2017

Peripheral Lipopolysaccharide Challenge Induces Long-Term Changes in Tyrosine Hydroxylase Regulation in the Adrenal Medulla.

scientific article

Rapamycin reduces motivated responding for cocaine and alters GluA1 expression in the ventral but not dorsal striatum.

scientific article published on 12 May 2016

Reconsidering the role of glial cells in chronic stress-induced dopaminergic neurons loss within the substantia nigra? Friend or foe?

scientific article published on 4 October 2016

Signal transduction pathways and tyrosine hydroxylase regulation in the adrenal medulla following glucoprivation: an in vivo analysis.

scientific article published on 21 May 2010

Spatiotemporal analysis of impaired microglia process movement at sites of secondary neurodegeneration post-stroke

scientific article published on 11 September 2018

Sustained administration of corticosterone at stress-like levels after stroke suppressed glial reactivity at sites of thalamic secondary neurodegeneration

scientific article published on 18 November 2017

The Effect of Social Defeat on Tyrosine Hydroxylase Phosphorylation in the Rat Brain and Adrenal Gland

scientific article published on 02 September 2010

The effects of footshock and immobilization stress on tyrosine hydroxylase phosphorylation in the rat locus coeruleus and adrenal gland

scientific article published on 14 July 2011

The sustained phase of tyrosine hydroxylase activation in vivo

scientific article published on 9 June 2012

Tyrosine hydroxylase phosphorylation in catecholaminergic brain regions: a marker of activation following acute hypotension and glucoprivation

scientific article

Tyrosine hydroxylase regulation in adult rat striatum following short-term neonatal exposure to manganese

scientific article published on 21 January 2016

Visual discrimination impairment after experimental stroke is associated with disturbances in the polarization of the astrocytic aquaporin-4 and increased accumulation of neurotoxic proteins

scientific article published on 09 May 2019

mTORC1 inhibition in the nucleus accumbens 'protects' against the expression of drug seeking and 'relapse' and is associated with reductions in GluA1 AMPAR and CAMKIIα levels.

scientific article

List of works by Lin Kooi Ong

Authors' response re: "Reconsidering the role of glial cells in chronic stress-induced dopaminergic neurons loss within the substantia nigra? Friend of foe?" by Ong et al. Brain Behavior and Immunity, 2016.

Chronic stress exposure following photothrombotic stroke is associated with increased levels of Amyloid beta accumulation and altered oligomerisation at sites of thalamic secondary neurodegeneration in mice

Chronic stress induced disruption of the peri-infarct neurovascular unit following experimentally induced photothrombotic stroke.

Early life peripheral lipopolysaccharide challenge reprograms catecholaminergic neurons

Early life stress and post-weaning high fat diet alter tyrosine hydroxylase regulation and AT1 receptor expression in the adrenal gland in a sex dependent manner.

Functional programming of the autonomic nervous system by early life immune exposure: implications for anxiety.

Growth Hormone Deficiency Is Frequent After Recent Stroke

Growth Hormone Promotes Motor Function after Experimental Stroke and Enhances Recovery-Promoting Mechanisms within the Peri-Infarct Area

Growth Hormone Treatment Promotes Remote Hippocampal Plasticity after Experimental Cortical Stroke

Impaired microglia process dynamics post-stroke are specific to sites of secondary neurodegeneration.

Increased microglial activation in the rat brain following neonatal exposure to a bacterial mimetic

Neonatal overfeeding increases capacity for catecholamine biosynthesis from the adrenal gland acutely and long-term in the male rat.

Neurobiological consequences of acute footshock stress: effects on tyrosine hydroxylase phosphorylation and activation in the rat brain and adrenal medulla.

Neurological impairment caused by Schistosoma mansoni systemic infection exhibits early features of idiopathic neurodegenerative disease

Oral administration of corticosterone at stress-like levels drives microglial but not vascular disturbances post-stroke

Peripheral Lipopolysaccharide Challenge Induces Long-Term Changes in Tyrosine Hydroxylase Regulation in the Adrenal Medulla.

Rapamycin reduces motivated responding for cocaine and alters GluA1 expression in the ventral but not dorsal striatum.

Reconsidering the role of glial cells in chronic stress-induced dopaminergic neurons loss within the substantia nigra? Friend or foe?

Signal transduction pathways and tyrosine hydroxylase regulation in the adrenal medulla following glucoprivation: an in vivo analysis.

Spatiotemporal analysis of impaired microglia process movement at sites of secondary neurodegeneration post-stroke

Sustained administration of corticosterone at stress-like levels after stroke suppressed glial reactivity at sites of thalamic secondary neurodegeneration

The Effect of Social Defeat on Tyrosine Hydroxylase Phosphorylation in the Rat Brain and Adrenal Gland

The effects of footshock and immobilization stress on tyrosine hydroxylase phosphorylation in the rat locus coeruleus and adrenal gland

The sustained phase of tyrosine hydroxylase activation in vivo

Tyrosine hydroxylase phosphorylation in catecholaminergic brain regions: a marker of activation following acute hypotension and glucoprivation

Tyrosine hydroxylase regulation in adult rat striatum following short-term neonatal exposure to manganese

Visual discrimination impairment after experimental stroke is associated with disturbances in the polarization of the astrocytic aquaporin-4 and increased accumulation of neurotoxic proteins

mTORC1 inhibition in the nucleus accumbens 'protects' against the expression of drug seeking and 'relapse' and is associated with reductions in GluA1 AMPAR and CAMKIIα levels.