List of works - Hind Lal

A novel cardioprotective p38-MAPK/mTOR pathway ⬇️

scientific article published on October 2, 2011

Anthrax lethal toxin induces acute diastolic dysfunction in rats through disruption of the phospholamban signaling network

scientific article

Cadherin-11 blockade reduces inflammation-driven fibrotic remodeling and improves outcomes after myocardial infarction

scientific article published on 19 September 2019

Cancer genetics and the cardiotoxicity of the therapeutics.

scientific article published on January 2013

Cardiac fibroblast glycogen synthase kinase-3β regulates ventricular remodeling and dysfunction in ischemic heart

scientific article published on 4 June 2014

Cardiomyocyte-GSK-3α promotes mPTP opening and heart failure in mice with chronic pressure overload

scientific article published on 27 March 2019

Cardiomyocyte-specific deletion of GSK-3β leads to cardiac dysfunction in a diet induced obesity model.

scientific article published on 2 February 2018

Cardiomyocyte-specific deletion of Gsk3α mitigates post-myocardial infarction remodeling, contractile dysfunction, and heart failure

scientific article

Caveolin and β1-integrin coordinate angiotensinogen expression in cardiac myocytes

scientific article published on 9 October 2012

Deletion of Cardiomyocyte Glycogen Synthase Kinase-3 Beta (GSK-3β) Improves Systemic Glucose Tolerance with Maintained Heart Function in Established Obesity

scientific article published on 30 April 2020

Entanglement of GSK-3β, β-catenin and TGF-β1 signaling network to regulate myocardial fibrosis.

scientific article

GSK-3alpha directly regulates beta-adrenergic signaling and the response of the heart to hemodynamic stress in mice

scientific journal article

GSK-3α is a central regulator of age-related pathologies in mice

scientific article

Glycogen synthase kinase-3beta regulates post-myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo

scientific article

Glycogen synthase kinase-3α limits ischemic injury, cardiac rupture, post-myocardial infarction remodeling and death

scientific article

Lethal and edema toxins of anthrax induce distinct hemodynamic dysfunction

scientific article published on 01 May 2007

Loss of Adult Cardiac Myocyte GSK-3 Leads to Mitotic Catastrophe Resulting in Fatal Dilated Cardiomyopathy.

scientific article published on 14 March 2016

Mechanistic Insights of Empagliflozin-Mediated Cardiac Benefits: Nearing the Starting Line : Editorial to: "Empagliflozin Improves Left Ventricular Diastolic Dysfunction in a Genetic Model of Type 2 Diabetes" by N. Hammoudi et al.

scientific article published on 21 July 2017

Mouse Models of Heart Failure with Preserved or Reduced Ejection Fraction

scientific article published on 25 April 2020

Prevention of liver cancer cachexia-induced cardiac wasting and heart failure

scientific article published on 29 August 2013

Rac1 and RhoA differentially regulate angiotensinogen gene expression in stretched cardiac fibroblasts

scientific journal article

Sorafenib cardiotoxicity increases mortality after myocardial infarction.

scientific article

The GSK-3 family as therapeutic target for myocardial diseases ⬇️

scientific article

List of works by Hind Lal

A novel cardioprotective p38-MAPK/mTOR pathway ⬇️

Anthrax lethal toxin induces acute diastolic dysfunction in rats through disruption of the phospholamban signaling network

Cadherin-11 blockade reduces inflammation-driven fibrotic remodeling and improves outcomes after myocardial infarction

Cancer genetics and the cardiotoxicity of the therapeutics.

Cardiac fibroblast glycogen synthase kinase-3β regulates ventricular remodeling and dysfunction in ischemic heart

Cardiomyocyte-GSK-3α promotes mPTP opening and heart failure in mice with chronic pressure overload

Cardiomyocyte-specific deletion of GSK-3β leads to cardiac dysfunction in a diet induced obesity model.

Cardiomyocyte-specific deletion of Gsk3α mitigates post-myocardial infarction remodeling, contractile dysfunction, and heart failure

Caveolin and β1-integrin coordinate angiotensinogen expression in cardiac myocytes

Deletion of Cardiomyocyte Glycogen Synthase Kinase-3 Beta (GSK-3β) Improves Systemic Glucose Tolerance with Maintained Heart Function in Established Obesity

Entanglement of GSK-3β, β-catenin and TGF-β1 signaling network to regulate myocardial fibrosis.

GSK-3alpha directly regulates beta-adrenergic signaling and the response of the heart to hemodynamic stress in mice

GSK-3α is a central regulator of age-related pathologies in mice

Glycogen synthase kinase-3beta regulates post-myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo

Glycogen synthase kinase-3α limits ischemic injury, cardiac rupture, post-myocardial infarction remodeling and death

Lethal and edema toxins of anthrax induce distinct hemodynamic dysfunction

Loss of Adult Cardiac Myocyte GSK-3 Leads to Mitotic Catastrophe Resulting in Fatal Dilated Cardiomyopathy.

Mechanistic Insights of Empagliflozin-Mediated Cardiac Benefits: Nearing the Starting Line : Editorial to: "Empagliflozin Improves Left Ventricular Diastolic Dysfunction in a Genetic Model of Type 2 Diabetes" by N. Hammoudi et al.

Mouse Models of Heart Failure with Preserved or Reduced Ejection Fraction

Prevention of liver cancer cachexia-induced cardiac wasting and heart failure

Rac1 and RhoA differentially regulate angiotensinogen gene expression in stretched cardiac fibroblasts

Sorafenib cardiotoxicity increases mortality after myocardial infarction.

The GSK-3 family as therapeutic target for myocardial diseases ⬇️

Troponin I-Interacting Protein Kinase