List of works - Johanna M.M. van Oers

A functional cancer genomics screen identifies a druggable synthetic lethal interaction between MSH3 and PRKDC.

scientific article published on 20 February 2014

A simple and fast method for the simultaneous detection of nine fibroblast growth factor receptor 3 mutations in bladder cancer and voided urine

scientific article published on 01 November 2005

Chromosome 9 deletions are more frequent than FGFR3 mutations in flat urothelial hyperplasias of the bladder.

scientific article

FGFR3 mutations and a normal CK20 staining pattern define low-grade noninvasive urothelial bladder tumours.

scientific article published on 12 January 2007

FGFR3 mutations in seborrheic keratoses are already present in flat lesions and associated with age and localization

scientific article published on 22 June 2007

FGFR3 mutations indicate better survival in invasive upper urinary tract and bladder tumours.

scientific article

Fibroblast growth factor receptor 3 mutations in bladder tumors correlate with low frequency of chromosome alterations

scientific article

High frequency of FGFR3 mutations in adenoid seborrheic keratoses.

scientific article

Induction of CAMEL/NY-ESO-ORF2-specific CD8+ T cells upon stimulation with dendritic cells infected with a modified Ad5 vector expressing a chimeric Ad5/35 fiber

scientific article published on March 2004

Low frequency of molecular changes and tumor recurrence in inverted papillomas of the urinary tract.

scientific article published on June 2007

Minimal PU.1 reduction induces a preleukemic state and promotes development of acute myeloid leukemia.

scientific article published on 7 September 2015

Minimal Reduction of PU.1 Is Sufficient to Induce a Preleukemic State and Promote Development of Acute Myeloid Leukemia ⬇️

scholarly article

Mosaicism of activating FGFR3 mutations in human skin causes epidermal nevi

scientific article

PMS2 endonuclease activity has distinct biological functions and is essential for genome maintenance

scientific article

Prediction of Progression of Non–Muscle-Invasive Bladder Cancer by WHO 1973 and 2004 Grading and by FGFR3 Mutation Status: A Prospective Study

article

The ATPase activity of MLH1 is required to orchestrate DNA double-strand breaks and end processing during class switch recombination

scientific article published on 26 March 2012

The absence of BRAF, FGFR3, and PIK3CA mutations differentiates lentigo simplex from melanocytic nevus and solar lentigo

scientific article published on 18 June 2009

[Correlation of FGFR3 mutations and chromosomal alterations in bladder cancer]

scientific article published on 01 January 2006

[Urothelial neoplasms in individuals younger than 20 years show very few genetic alterations and have a favourable clinical outcome]

scientific article published on 01 January 2006

List of works by Johanna M.M. van Oers

A functional cancer genomics screen identifies a druggable synthetic lethal interaction between MSH3 and PRKDC.

A simple and fast method for the simultaneous detection of nine fibroblast growth factor receptor 3 mutations in bladder cancer and voided urine

Chromosome 9 deletions are more frequent than FGFR3 mutations in flat urothelial hyperplasias of the bladder.

FGFR3 mutations and a normal CK20 staining pattern define low-grade noninvasive urothelial bladder tumours.

FGFR3 mutations in seborrheic keratoses are already present in flat lesions and associated with age and localization

FGFR3 mutations indicate better survival in invasive upper urinary tract and bladder tumours.

Fibroblast growth factor receptor 3 mutations in bladder tumors correlate with low frequency of chromosome alterations

High frequency of FGFR3 mutations in adenoid seborrheic keratoses.

Induction of CAMEL/NY-ESO-ORF2-specific CD8+ T cells upon stimulation with dendritic cells infected with a modified Ad5 vector expressing a chimeric Ad5/35 fiber

Low frequency of molecular changes and tumor recurrence in inverted papillomas of the urinary tract.

Minimal PU.1 reduction induces a preleukemic state and promotes development of acute myeloid leukemia.

Minimal Reduction of PU.1 Is Sufficient to Induce a Preleukemic State and Promote Development of Acute Myeloid Leukemia ⬇️

Mosaicism of activating FGFR3 mutations in human skin causes epidermal nevi

PMS2 endonuclease activity has distinct biological functions and is essential for genome maintenance

Prediction of Progression of Non–Muscle-Invasive Bladder Cancer by WHO 1973 and 2004 Grading and by FGFR3 Mutation Status: A Prospective Study

The ATPase activity of MLH1 is required to orchestrate DNA double-strand breaks and end processing during class switch recombination

The absence of BRAF, FGFR3, and PIK3CA mutations differentiates lentigo simplex from melanocytic nevus and solar lentigo

[Correlation of FGFR3 mutations and chromosomal alterations in bladder cancer]

[Urothelial neoplasms in individuals younger than 20 years show very few genetic alterations and have a favourable clinical outcome]