List of works - Naomi Goldfinger

A novel translocation breakpoint within the BPTF gene is associated with a pre-malignant phenotype

scientific article

Cancer therapeutic approach based on conformational stabilization of mutant p53 protein by small peptides.

scientific article

Epigenetic polymorphism and the stochastic formation of differentially methylated regions in normal and cancerous tissues

scientific article published on 14 October 2012

Inactivation of myocardin and p16 during malignant transformation contributes to a differentiation defect.

scientific article published in February 2007

Modulated expression of WFDC1 during carcinogenesis and cellular senescence

scientific article published on 08 October 2008

Mutant p53 facilitates somatic cell reprogramming and augments the malignant potential of reprogrammed cells

scientific article published on 9 August 2010

Mutant p53 gain of function underlies high expression levels of colorectal cancer stem cells markers.

scientific article published on 18 January 2018

Mutant p53 protects cells from 12-O-tetradecanoylphorbol-13-acetate-induced death by attenuating activating transcription factor 3 induction

scientific article published on November 2006

Prostate stromal cells produce CXCL-1, CXCL-2, CXCL-3 and IL-8 in response to epithelia-secreted IL-1.

scientific article published on 20 February 2009

SPATA18, a spermatogenesis-associated gene, is a novel transcriptional target of p53 and p63

scientific journal article

TMPRSS2/ERG promotes epithelial to mesenchymal transition through the ZEB1/ZEB2 axis in a prostate cancer model

scientific article

Transcriptional activity of ATF3 in the stromal compartment of tumors promotes cancer progression.

scientific article

Various p53 mutant proteins differently regulate the Ras circuit to induce a cancer-related gene signature

scientific article

hTERT-immortalized prostate epithelial and stromal-derived cells: an authentic in vitro model for differentiation and carcinogenesis

scientific article

p53 Regulates the Ras circuit to inhibit the expression of a cancer-related gene signature by various molecular pathways.

scientific article published on 2 March 2010

p53-Repressed miRNAs are involved with E2F in a feed-forward loop promoting proliferation.

scientific article

p53-dependent transcriptional regulation of EDA2R and its involvement in chemotherapy-induced hair loss

scientific journal article

List of works by Naomi Goldfinger

A novel translocation breakpoint within the BPTF gene is associated with a pre-malignant phenotype

Cancer therapeutic approach based on conformational stabilization of mutant p53 protein by small peptides.

Epigenetic polymorphism and the stochastic formation of differentially methylated regions in normal and cancerous tissues

Inactivation of myocardin and p16 during malignant transformation contributes to a differentiation defect.

Modulated expression of WFDC1 during carcinogenesis and cellular senescence

Mutant p53 facilitates somatic cell reprogramming and augments the malignant potential of reprogrammed cells

Mutant p53 gain of function underlies high expression levels of colorectal cancer stem cells markers.

Mutant p53 protects cells from 12-O-tetradecanoylphorbol-13-acetate-induced death by attenuating activating transcription factor 3 induction

Prostate stromal cells produce CXCL-1, CXCL-2, CXCL-3 and IL-8 in response to epithelia-secreted IL-1.

SPATA18, a spermatogenesis-associated gene, is a novel transcriptional target of p53 and p63

TMPRSS2/ERG promotes epithelial to mesenchymal transition through the ZEB1/ZEB2 axis in a prostate cancer model

Transcriptional activity of ATF3 in the stromal compartment of tumors promotes cancer progression.

Various p53 mutant proteins differently regulate the Ras circuit to induce a cancer-related gene signature

hTERT-immortalized prostate epithelial and stromal-derived cells: an authentic in vitro model for differentiation and carcinogenesis

p53 Regulates the Ras circuit to inhibit the expression of a cancer-related gene signature by various molecular pathways.

p53-Repressed miRNAs are involved with E2F in a feed-forward loop promoting proliferation.

p53-dependent transcriptional regulation of EDA2R and its involvement in chemotherapy-induced hair loss